Search Results - "Al-Riyami, Lamyaa"

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The role of individual protein kinase C isoforms in mouse mast cell function and their targeting by the immunomodulatory parasitic worm product, ES-62.

Authors: Bell, Kara S. Al-Riyami, Lamyaa Lumb, Felicity E. et al.

Source: Immunology Letters. Nov2015, Vol. 168 Issue 1, p31-40. 10p.

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An investigation of the ability of the secreted filarial nematode product ES-62 to inhibit TH1 responses to heterologous molecules

Authors: Al-Riyami, Lamyaa Rashid

URL: http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.443123

Prophylactic and therapeutic treatment with a synthetic analogue of a parasitic worm product prevents experimental arthritis and inhibits IL-1β production via NRF2-mediated counter-regulation of the inflammasome.

Authors: Rzepecka, Justyna Pineda, Miguel A. Al-Riyami, Lamyaa et al.

Source: Journal of Autoimmunity. Jun2015, Vol. 60, p59-73. 15p.

ES-62 Protects Against Collagen-Induced Arthritis by Resetting Interleukin-22 Toward Resolution of Inflammation in the Joints.

Authors: Pineda, Miguel A. Rodgers, David T. Al‐Riyami, Lamyaa et al.

Source: Arthritis & Rheumatology. Jun2014, Vol. 66 Issue 6, p1492-1503. 12p.

Protection against collagen-induced arthritis in mice afforded by the parasitic worm product, ES-62, is associated with restoration of the levels of interleukin-10-producing B cells and reduced plasma cell infiltration of the joints.

Authors: Rodgers, David T. Pineda, Miguel A. McGrath, Mairi A. et al.

Source: Immunology. Mar2014, Vol. 141 Issue 3, p457-466. 10p.

Mast Cell Subsets and Their Functional Modulation by the Acanthocheilonema viteae Product ES-62.

Authors: Ball, Dimity H. Tay, Hwee Kee Bell, Kara S. et al.

Source: Journal of Parasitology Research. 2013, p1-13. 13p.

The helminth product ES-62 protects against septic shock via Toll-like receptor 4-dependent autophagosomal degradation of the adaptor MyD88.

Authors: Puneet, Padmam McGrath, Mairi A Tay, Hwee Kee et al.

Source: Nature Immunology. Apr2011, Vol. 12 Issue 4, p344-351. 8p. 7 Color Photographs, 1 Black and White Photograph.

The impact of anthelmintic drugs on weight gain of smallholder goats in subtropical regions.

Authors: Bessell, Paul R. Sargison, Neil D. Mirende, Kichuki et al.

Source: Preventive Veterinary Medicine. Nov2018, Vol. 159, p72-81. 10p.

Assessing the impact of a novel strategy for delivering animal health interventions to smallholder farmers.

Authors: Bessell, Paul Richard Kushwaha, Peetambar Mosha, Roggers et al.

Source: Preventive Veterinary Medicine. Nov2017, Vol. 147, p108-116. 9p.

Failure of the Anti-Inflammatory Parasitic Worm Product ES-62 to Provide Protection in Mouse Models of Type I Diabetes, Multiple Sclerosis, and Inflammatory Bowel Disease.

Authors: Doonan, James Thomas, David Wong, Michelle H. et al.

Source: Molecules. Oct2018, Vol. 23 Issue 10, p2669. 1p. 3 Diagrams, 4 Graphs.

The Parasitic Worm Product ES-62 Targets Myeloid Differentiation Factor 88-Dependent Effector Mechanisms to Suppress Antinuclear Antibody Production and Proteinuria in MRL/ lpr Mice.

Authors: Rodgers, David T. McGrath, Mairi A. Pineda, Miguel A. et al.

Source: Arthritis & Rheumatology. Apr2015, Vol. 67 Issue 4, p1023-1035. 13p.

The parasitic helminth product ES-62 suppresses pathogenesis in collagen-induced arthritis by targeting the interleukin-17-producing cellular network at multiple sites.

Authors: Pineda, Miguel A. Mcgrath, Mairi A. Smith, Pauline C. et al.

Source: Arthritis & Rheumatism. Oct2012, Vol. 64 Issue 10, p3168-3178. 11p.

The helminth product, ES-62 modulates dendritic cell responses by inducing the selective autophagolysosomal degradation of TLR-transducers, as exemplified by PKCδ.

Authors: Eason, Russell J. Bell, Kara S. Marshall, Fraser A. et al.

Source: Scientific Reports. 11/25/2016, p37276. 1p.

An analysis of the impact of Newcastle disease vaccination and husbandry practice on smallholder chicken productivity in Uganda.

Authors: Bessell, Paul R Woolley, Roy Stevenson, Stuart et al.

Source: Preventive Veterinary Medicine. Apr2020, Vol. 177, pN.PAG-N.PAG. 1p.