Search Results - "Al-Riyami, Lamyaa"

The helminth product ES-62 protects against septic shock via Toll-like receptor 4-dependent autophagosomal degradation of the adaptor MyD88.

Authors: Puneet, Padmam McGrath, Mairi A Tay, Hwee Kee et al.

Source: Nature Immunology. Aug2011, Vol. 12 Issue 8, p804-804. 1p.

Protection against collagen-induced arthritis in mice afforded by the parasitic worm product, ES-62, is associated with restoration of the levels of interleukin-10-producing B cells and reduced plasma cell infiltration of the joints.

Authors: Rodgers, David T. Pineda, Miguel A. McGrath, Mairi A. et al.

Source: Immunology. Mar2014, Vol. 141 Issue 3, p457-466. 10p.

Mast Cell Subsets and Their Functional Modulation by the Acanthocheilonema viteae Product ES-62.

Authors: Ball, Dimity H. Tay, Hwee Kee Bell, Kara S. et al.

Source: Journal of Parasitology Research. 2013, p1-13. 13p.

The helminth product ES-62 protects against septic shock via Toll-like receptor 4-dependent autophagosomal degradation of the adaptor MyD88.

Authors: Puneet, Padmam McGrath, Mairi A Tay, Hwee Kee et al.

Source: Nature Immunology. Apr2011, Vol. 12 Issue 4, p344-351. 8p. 7 Color Photographs, 1 Black and White Photograph.

Failure of the Anti-Inflammatory Parasitic Worm Product ES-62 to Provide Protection in Mouse Models of Type I Diabetes, Multiple Sclerosis, and Inflammatory Bowel Disease.

Authors: Doonan, James Thomas, David Wong, Michelle H. et al.

Source: Molecules. Oct2018, Vol. 23 Issue 10, p2669. 1p. 3 Diagrams, 4 Graphs.

The helminth product, ES-62 modulates dendritic cell responses by inducing the selective autophagolysosomal degradation of TLR-transducers, as exemplified by PKCδ.

Authors: Eason, Russell J. Bell, Kara S. Marshall, Fraser A. et al.

Source: Scientific Reports. 11/25/2016, p37276. 1p.