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Antisense inhibition of connective tissue growth factor (CTGF/CCN2) mRNA limits hypertrophic scarring without affecting wound healing in vivo.

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Title: Antisense inhibition of connective tissue growth factor (CTGF/CCN2) mRNA limits hypertrophic scarring without affecting wound healing in vivo.
Authors: Sisco M (AUTHOR), Kryger ZB (AUTHOR), O'Shaughnessy KD (AUTHOR), Kim PS (AUTHOR), Schultz GS (AUTHOR), Ding X (AUTHOR), Roy NK (AUTHOR), Dean NM (AUTHOR), Mustoe TA (AUTHOR)
Source: Wound Repair & Regeneration. Sep/Oct2008, Vol. 16 Issue 5, p661-673. 13p.
URL: 34137876
Database: Academic Search Ultimate
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Header DbId: asn
DbLabel: Academic Search Ultimate
An: 105676118
AccessLevel: 2
PubType: Academic Journal
PubTypeId: academicJournal
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  Data: Antisense inhibition of connective tissue growth factor (CTGF/CCN2) mRNA limits hypertrophic scarring without affecting wound healing in vivo.
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  Data: <searchLink fieldCode="JN" term="%22Wound+Repair+%26+Regeneration%22">Wound Repair & Regeneration</searchLink>. Sep/Oct2008, Vol. 16 Issue 5, p661-673. 13p.
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RecordInfo BibRecord:
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      – Type: doi
        Value: 10.1111/j.1524-475x.2008.00416.x
    Languages:
      – Code: eng
        Text: English
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      Pagination:
        PageCount: 13
        StartPage: 661
    Titles:
      – TitleFull: Antisense inhibition of connective tissue growth factor (CTGF/CCN2) mRNA limits hypertrophic scarring without affecting wound healing in vivo.
        Type: main
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          Name:
            NameFull: Sisco M
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            NameFull: Kryger ZB
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            NameFull: O'Shaughnessy KD
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            NameFull: Kim PS
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            NameFull: Schultz GS
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            NameFull: Ding X
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            NameFull: Roy NK
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            NameFull: Dean NM
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            NameFull: Mustoe TA
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            – D: 01
              M: 09
              Text: Sep/Oct2008
              Type: published
              Y: 2008
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              Value: 10671927
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              Value: 16
            – Type: issue
              Value: 5
          Titles:
            – TitleFull: Wound Repair & Regeneration
              Type: main
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