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Beta-cell deficit due to increased apoptosis in the human islet amyloid polypeptide transgenic (HIP) rat recapitulates the metabolic defects present in type 2 diabetes.

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Title: Beta-cell deficit due to increased apoptosis in the human islet amyloid polypeptide transgenic (HIP) rat recapitulates the metabolic defects present in type 2 diabetes.
Authors: Matveyenko AV (AUTHOR), Butler PC (AUTHOR)
Source: Diabetes. Jul2006, Vol. 55 Issue 7, p2106-2114. 9p.
URL: 21601365
Database: Academic Search Ultimate
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  Data: Beta-cell deficit due to increased apoptosis in the human islet amyloid polypeptide transgenic (HIP) rat recapitulates the metabolic defects present in type 2 diabetes.
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  Data: <searchLink fieldCode="AR" term="%22Matveyenko+AV%22">Matveyenko AV</searchLink> (AUTHOR)<br /><searchLink fieldCode="AR" term="%22Butler+PC%22">Butler PC</searchLink> (AUTHOR)
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  Data: <searchLink fieldCode="JN" term="%22Diabetes%22">Diabetes</searchLink>. Jul2006, Vol. 55 Issue 7, p2106-2114. 9p.
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      – Type: doi
        Value: 10.2337/db05-1672
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      – Code: eng
        Text: English
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        PageCount: 9
        StartPage: 2106
    Titles:
      – TitleFull: Beta-cell deficit due to increased apoptosis in the human islet amyloid polypeptide transgenic (HIP) rat recapitulates the metabolic defects present in type 2 diabetes.
        Type: main
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            NameFull: Matveyenko AV
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            NameFull: Butler PC
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            – D: 01
              M: 07
              Text: Jul2006
              Type: published
              Y: 2006
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              Value: 00121797
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              Value: 55
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              Value: 7
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            – TitleFull: Diabetes
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