APA (7th ed.) Citation

Hridi, S. U., Franssen, A. J., Jiang, H., & Bushell, T. J. (2019). Interleukin-16 inhibits sodium channel function and GluA1 phosphorylation via CD4- and CD9-independent mechanisms to reduce hippocampal neuronal excitability and synaptic activity. MCN: Molecular & Cellular Neuroscience, 95, 71. https://doi.org/10.1016/j.mcn.2019.01.002

Chicago Style (17th ed.) Citation

Hridi, Shehla U., Aimée J.P.M Franssen, Hui-Rong Jiang, and Trevor J. Bushell. "Interleukin-16 Inhibits Sodium Channel Function and GluA1 Phosphorylation via CD4- and CD9-independent Mechanisms to Reduce Hippocampal Neuronal Excitability and Synaptic Activity." MCN: Molecular & Cellular Neuroscience 95 (2019): 71. https://doi.org/10.1016/j.mcn.2019.01.002.

MLA (9th ed.) Citation

Hridi, Shehla U., et al. "Interleukin-16 Inhibits Sodium Channel Function and GluA1 Phosphorylation via CD4- and CD9-independent Mechanisms to Reduce Hippocampal Neuronal Excitability and Synaptic Activity." MCN: Molecular & Cellular Neuroscience, vol. 95, 2019, p. 71, https://doi.org/10.1016/j.mcn.2019.01.002.