Loss of hepatic aldolase B activates Akt and promotes hepatocellular carcinogenesis by destabilizing the Aldob/Akt/PP2A protein complex.
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| Title: | Loss of hepatic aldolase B activates Akt and promotes hepatocellular carcinogenesis by destabilizing the Aldob/Akt/PP2A protein complex. |
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| Authors: | He, Xuxiao1,2 (AUTHOR), Li, Min1,2 (AUTHOR), Yu, Hongming3 (AUTHOR), Liu, Guijun1,2 (AUTHOR), Wang, Ningning1,2 (AUTHOR), Yin, Chunzhao2,4 (AUTHOR), Tu, Qiaochu2,4 (AUTHOR), Narla, Goutham5 (AUTHOR), Tao, Yongzhen1 (AUTHOR) yztao01@sibs.ac.cn, Cheng, Shuqun3 (AUTHOR) yztao01@sibs.ac.cn, Yin, Huiyong1,2,4,6 (AUTHOR) yztao01@sibs.ac.cn |
| Source: | PLoS Biology. 12/4/2020, Vol. 18 Issue 12, p1-31. 31p. 2 Color Photographs, 4 Black and White Photographs, 1 Diagram, 1 Graph. |
| Database: | Academic Search Ultimate |
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| Header | DbId: asn DbLabel: Academic Search Ultimate An: 147380196 AccessLevel: 2 PubType: Academic Journal PubTypeId: academicJournal PreciseRelevancyScore: 0 |
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| Items | – Name: Title Label: Title Group: Ti Data: Loss of hepatic aldolase B activates Akt and promotes hepatocellular carcinogenesis by destabilizing the Aldob/Akt/PP2A protein complex. – Name: Author Label: Authors Group: Au Data: <searchLink fieldCode="AR" term="%22He%2C+Xuxiao%22">He, Xuxiao</searchLink><relatesTo>1,2</relatesTo> (AUTHOR)<br /><searchLink fieldCode="AR" term="%22Li%2C+Min%22">Li, Min</searchLink><relatesTo>1,2</relatesTo> (AUTHOR)<br /><searchLink fieldCode="AR" term="%22Yu%2C+Hongming%22">Yu, Hongming</searchLink><relatesTo>3</relatesTo> (AUTHOR)<br /><searchLink fieldCode="AR" term="%22Liu%2C+Guijun%22">Liu, Guijun</searchLink><relatesTo>1,2</relatesTo> (AUTHOR)<br /><searchLink fieldCode="AR" term="%22Wang%2C+Ningning%22">Wang, Ningning</searchLink><relatesTo>1,2</relatesTo> (AUTHOR)<br /><searchLink fieldCode="AR" term="%22Yin%2C+Chunzhao%22">Yin, Chunzhao</searchLink><relatesTo>2,4</relatesTo> (AUTHOR)<br /><searchLink fieldCode="AR" term="%22Tu%2C+Qiaochu%22">Tu, Qiaochu</searchLink><relatesTo>2,4</relatesTo> (AUTHOR)<br /><searchLink fieldCode="AR" term="%22Narla%2C+Goutham%22">Narla, Goutham</searchLink><relatesTo>5</relatesTo> (AUTHOR)<br /><searchLink fieldCode="AR" term="%22Tao%2C+Yongzhen%22">Tao, Yongzhen</searchLink><relatesTo>1</relatesTo> (AUTHOR)<i> yztao01@sibs.ac.cn</i><br /><searchLink fieldCode="AR" term="%22Cheng%2C+Shuqun%22">Cheng, Shuqun</searchLink><relatesTo>3</relatesTo> (AUTHOR)<i> yztao01@sibs.ac.cn</i><br /><searchLink fieldCode="AR" term="%22Yin%2C+Huiyong%22">Yin, Huiyong</searchLink><relatesTo>1,2,4,6</relatesTo> (AUTHOR)<i> yztao01@sibs.ac.cn</i> – Name: TitleSource Label: Source Group: Src Data: <searchLink fieldCode="JN" term="%22PLoS+Biology%22">PLoS Biology</searchLink>. 12/4/2020, Vol. 18 Issue 12, p1-31. 31p. 2 Color Photographs, 4 Black and White Photographs, 1 Diagram, 1 Graph. |
| PLink | https://search.ebscohost.com/login.aspx?direct=true&site=eds-live&db=asn&AN=147380196 |
| RecordInfo | BibRecord: BibEntity: Identifiers: – Type: doi Value: 10.1371/journal.pbio.3000803 Languages: – Code: eng Text: English PhysicalDescription: Pagination: PageCount: 31 StartPage: 1 Titles: – TitleFull: Loss of hepatic aldolase B activates Akt and promotes hepatocellular carcinogenesis by destabilizing the Aldob/Akt/PP2A protein complex. Type: main BibRelationships: HasContributorRelationships: – PersonEntity: Name: NameFull: He, Xuxiao – PersonEntity: Name: NameFull: Li, Min – PersonEntity: Name: NameFull: Yu, Hongming – PersonEntity: Name: NameFull: Liu, Guijun – PersonEntity: Name: NameFull: Wang, Ningning – PersonEntity: Name: NameFull: Yin, Chunzhao – PersonEntity: Name: NameFull: Tu, Qiaochu – PersonEntity: Name: NameFull: Narla, Goutham – PersonEntity: Name: NameFull: Tao, Yongzhen – PersonEntity: Name: NameFull: Cheng, Shuqun – PersonEntity: Name: NameFull: Yin, Huiyong IsPartOfRelationships: – BibEntity: Dates: – D: 04 M: 12 Text: 12/4/2020 Type: published Y: 2020 Identifiers: – Type: issn-print Value: 15449173 Numbering: – Type: volume Value: 18 – Type: issue Value: 12 Titles: – TitleFull: PLoS Biology Type: main |
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