BSCL2/Seipin deficiency in hearts causes cardiac energy deficit and dysfunction via inducing excessive lipid catabolism.

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Title: BSCL2/Seipin deficiency in hearts causes cardiac energy deficit and dysfunction via inducing excessive lipid catabolism.
Authors: Zhou, Hongyi1 (AUTHOR), Li, Jie2 (AUTHOR), Su, Huabo2 (AUTHOR), Li, Ji3 (AUTHOR), Lydic, Todd A.4 (AUTHOR), Young, Martin E5 (AUTHOR), Chen, Weiqin1 (AUTHOR) wechen@augusta.edu
Source: Clinical & Translational Medicine. Apr2022, Vol. 12 Issue 4, p1-18. 18p.
Database: Academic Search Ultimate
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  Data: BSCL2/Seipin deficiency in hearts causes cardiac energy deficit and dysfunction via inducing excessive lipid catabolism.
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  Data: <searchLink fieldCode="AR" term="%22Zhou%2C+Hongyi%22">Zhou, Hongyi</searchLink><relatesTo>1</relatesTo> (AUTHOR)<br /><searchLink fieldCode="AR" term="%22Li%2C+Jie%22">Li, Jie</searchLink><relatesTo>2</relatesTo> (AUTHOR)<br /><searchLink fieldCode="AR" term="%22Su%2C+Huabo%22">Su, Huabo</searchLink><relatesTo>2</relatesTo> (AUTHOR)<br /><searchLink fieldCode="AR" term="%22Li%2C+Ji%22">Li, Ji</searchLink><relatesTo>3</relatesTo> (AUTHOR)<br /><searchLink fieldCode="AR" term="%22Lydic%2C+Todd+A%2E%22">Lydic, Todd A.</searchLink><relatesTo>4</relatesTo> (AUTHOR)<br /><searchLink fieldCode="AR" term="%22Young%2C+Martin+E%22">Young, Martin E</searchLink><relatesTo>5</relatesTo> (AUTHOR)<br /><searchLink fieldCode="AR" term="%22Chen%2C+Weiqin%22">Chen, Weiqin</searchLink><relatesTo>1</relatesTo> (AUTHOR)<i> wechen@augusta.edu</i>
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  Data: <searchLink fieldCode="JN" term="%22Clinical+%26+Translational+Medicine%22">Clinical & Translational Medicine</searchLink>. Apr2022, Vol. 12 Issue 4, p1-18. 18p.
PLink https://search.ebscohost.com/login.aspx?direct=true&site=eds-live&db=asn&AN=156617568
RecordInfo BibRecord:
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    Identifiers:
      – Type: doi
        Value: 10.1002/ctm2.736
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      – Code: eng
        Text: English
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        PageCount: 18
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      – TitleFull: BSCL2/Seipin deficiency in hearts causes cardiac energy deficit and dysfunction via inducing excessive lipid catabolism.
        Type: main
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            NameFull: Zhou, Hongyi
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            NameFull: Li, Jie
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            NameFull: Su, Huabo
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            NameFull: Li, Ji
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            NameFull: Lydic, Todd A.
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            NameFull: Young, Martin E
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            NameFull: Chen, Weiqin
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            – D: 01
              M: 04
              Text: Apr2022
              Type: published
              Y: 2022
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            – TitleFull: Clinical & Translational Medicine
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