Activation of CB2R by synthetic CB2R agonist, PM289, improves brain endothelial barrier properties, decreases inflammatory response and enhances endothelial repair.

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Title: Activation of CB2R by synthetic CB2R agonist, PM289, improves brain endothelial barrier properties, decreases inflammatory response and enhances endothelial repair.
Authors: Bullock, Trent A.1,2, Galpayage Dona, Kalpani N. Udeni1, Hale, Jonathan F.1, Morales, Paula3, Jagerovic, Nadine3, Andrews, Allison M.1,2, Ramirez, Servio H.1,2,4 servio@temple.edu
Source: NeuroImmune Pharmacology & Therapeutics. Dec2023, Vol. 2 Issue 4, p387-400. 14p.
Database: Academic Search Ultimate
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An: 174017807
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  Data: Activation of CB2R by synthetic CB2R agonist, PM289, improves brain endothelial barrier properties, decreases inflammatory response and enhances endothelial repair.
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  Data: <searchLink fieldCode="JN" term="%22NeuroImmune+Pharmacology+%26+Therapeutics%22">NeuroImmune Pharmacology & Therapeutics</searchLink>. Dec2023, Vol. 2 Issue 4, p387-400. 14p.
PLink https://search.ebscohost.com/login.aspx?direct=true&site=eds-live&db=asn&AN=174017807
RecordInfo BibRecord:
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    Identifiers:
      – Type: doi
        Value: 10.1515/nipt-2023-0016
    Languages:
      – Code: eng
        Text: English
    PhysicalDescription:
      Pagination:
        PageCount: 14
        StartPage: 387
    Titles:
      – TitleFull: Activation of CB2R by synthetic CB2R agonist, PM289, improves brain endothelial barrier properties, decreases inflammatory response and enhances endothelial repair.
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            NameFull: Bullock, Trent A.
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            NameFull: Galpayage Dona, Kalpani N. Udeni
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            NameFull: Hale, Jonathan F.
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            NameFull: Morales, Paula
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            NameFull: Jagerovic, Nadine
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            NameFull: Andrews, Allison M.
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            NameFull: Ramirez, Servio H.
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            – D: 01
              M: 12
              Text: Dec2023
              Type: published
              Y: 2023
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          Titles:
            – TitleFull: NeuroImmune Pharmacology & Therapeutics
              Type: main
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