APA (7th ed.) Citation

Dai, Z., Xie, B., Jiang, C., Peng, Y., Lin, J., Chen, Q., & Sun, J. (2025). Aerosolized nicotine-free e-liquid base constituents exacerbates mitochondrial dysfunction and endothelial glycocalyx shedding via the AKT/GSK3β-mPTP pathway in lung injury models. Respiratory Research, 26(1), 1. https://doi.org/10.1186/s12931-025-03155-3

Chicago Style (17th ed.) Citation

Dai, Ziyu, Bin Xie, Chen Jiang, Yun Peng, Jianing Lin, Qiong Chen, and Jingyi Sun. "Aerosolized Nicotine-free E-liquid Base Constituents Exacerbates Mitochondrial Dysfunction and Endothelial Glycocalyx Shedding via the AKT/GSK3β-mPTP Pathway in Lung Injury Models." Respiratory Research 26, no. 1 (2025): 1. https://doi.org/10.1186/s12931-025-03155-3.

MLA (9th ed.) Citation

Dai, Ziyu, et al. "Aerosolized Nicotine-free E-liquid Base Constituents Exacerbates Mitochondrial Dysfunction and Endothelial Glycocalyx Shedding via the AKT/GSK3β-mPTP Pathway in Lung Injury Models." Respiratory Research, vol. 26, no. 1, 2025, p. 1, https://doi.org/10.1186/s12931-025-03155-3.