Targeting the ERK1/2 and p38 MAPK pathways attenuates Golgi tethering factor golgin-97 depletion-induced cancer progression in breast cancer.
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| Title: | Targeting the ERK1/2 and p38 MAPK pathways attenuates Golgi tethering factor golgin-97 depletion-induced cancer progression in breast cancer. |
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| Authors: | Liu, Yu-Chin1 (AUTHOR), Lin, Tsung-Jen1,2 (AUTHOR), Chong, Kowit-Yu3,4,5,6 (AUTHOR), Chen, Guan-Ying1 (AUTHOR), Kuo, Chia-Yu1 (AUTHOR), Lin, Yi-Yun7 (AUTHOR), Chang, Chia-Wei7 (AUTHOR), Hsiao, Ting-Feng7 (AUTHOR), Wang, Chih-Liang8,9 (AUTHOR), Shih, Yo-Chen7 (AUTHOR), Yu, Chia-Jung1,7,9,10 (AUTHOR) yucj1124@mail.cgu.edu.tw |
| Source: | Cell Communication & Signaling. 3/3/2025, Vol. 23 Issue 1, p1-18. 18p. |
| Database: | Academic Search Ultimate |
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| FullText | Links: – Type: pdflink Text: Availability: 1 |
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| Header | DbId: asn DbLabel: Academic Search Ultimate An: 183405199 AccessLevel: 2 PubType: Academic Journal PubTypeId: academicJournal PreciseRelevancyScore: 0 |
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| Items | – Name: Title Label: Title Group: Ti Data: Targeting the ERK1/2 and p38 MAPK pathways attenuates Golgi tethering factor golgin-97 depletion-induced cancer progression in breast cancer. – Name: Author Label: Authors Group: Au Data: <searchLink fieldCode="AR" term="%22Liu%2C+Yu-Chin%22">Liu, Yu-Chin</searchLink><relatesTo>1</relatesTo> (AUTHOR)<br /><searchLink fieldCode="AR" term="%22Lin%2C+Tsung-Jen%22">Lin, Tsung-Jen</searchLink><relatesTo>1,2</relatesTo> (AUTHOR)<br /><searchLink fieldCode="AR" term="%22Chong%2C+Kowit-Yu%22">Chong, Kowit-Yu</searchLink><relatesTo>3,4,5,6</relatesTo> (AUTHOR)<br /><searchLink fieldCode="AR" term="%22Chen%2C+Guan-Ying%22">Chen, Guan-Ying</searchLink><relatesTo>1</relatesTo> (AUTHOR)<br /><searchLink fieldCode="AR" term="%22Kuo%2C+Chia-Yu%22">Kuo, Chia-Yu</searchLink><relatesTo>1</relatesTo> (AUTHOR)<br /><searchLink fieldCode="AR" term="%22Lin%2C+Yi-Yun%22">Lin, Yi-Yun</searchLink><relatesTo>7</relatesTo> (AUTHOR)<br /><searchLink fieldCode="AR" term="%22Chang%2C+Chia-Wei%22">Chang, Chia-Wei</searchLink><relatesTo>7</relatesTo> (AUTHOR)<br /><searchLink fieldCode="AR" term="%22Hsiao%2C+Ting-Feng%22">Hsiao, Ting-Feng</searchLink><relatesTo>7</relatesTo> (AUTHOR)<br /><searchLink fieldCode="AR" term="%22Wang%2C+Chih-Liang%22">Wang, Chih-Liang</searchLink><relatesTo>8,9</relatesTo> (AUTHOR)<br /><searchLink fieldCode="AR" term="%22Shih%2C+Yo-Chen%22">Shih, Yo-Chen</searchLink><relatesTo>7</relatesTo> (AUTHOR)<br /><searchLink fieldCode="AR" term="%22Yu%2C+Chia-Jung%22">Yu, Chia-Jung</searchLink><relatesTo>1,7,9,10</relatesTo> (AUTHOR)<i> yucj1124@mail.cgu.edu.tw</i> – Name: TitleSource Label: Source Group: Src Data: <searchLink fieldCode="JN" term="%22Cell+Communication+%26+Signaling%22">Cell Communication & Signaling</searchLink>. 3/3/2025, Vol. 23 Issue 1, p1-18. 18p. |
| PLink | https://search.ebscohost.com/login.aspx?direct=true&site=eds-live&db=asn&AN=183405199 |
| RecordInfo | BibRecord: BibEntity: Identifiers: – Type: doi Value: 10.1186/s12964-024-02010-0 Languages: – Code: eng Text: English PhysicalDescription: Pagination: PageCount: 18 StartPage: 1 Titles: – TitleFull: Targeting the ERK1/2 and p38 MAPK pathways attenuates Golgi tethering factor golgin-97 depletion-induced cancer progression in breast cancer. Type: main BibRelationships: HasContributorRelationships: – PersonEntity: Name: NameFull: Liu, Yu-Chin – PersonEntity: Name: NameFull: Lin, Tsung-Jen – PersonEntity: Name: NameFull: Chong, Kowit-Yu – PersonEntity: Name: NameFull: Chen, Guan-Ying – PersonEntity: Name: NameFull: Kuo, Chia-Yu – PersonEntity: Name: NameFull: Lin, Yi-Yun – PersonEntity: Name: NameFull: Chang, Chia-Wei – PersonEntity: Name: NameFull: Hsiao, Ting-Feng – PersonEntity: Name: NameFull: Wang, Chih-Liang – PersonEntity: Name: NameFull: Shih, Yo-Chen – PersonEntity: Name: NameFull: Yu, Chia-Jung IsPartOfRelationships: – BibEntity: Dates: – D: 03 M: 03 Text: 3/3/2025 Type: published Y: 2025 Identifiers: – Type: issn-print Value: 1478811X Numbering: – Type: volume Value: 23 – Type: issue Value: 1 Titles: – TitleFull: Cell Communication & Signaling Type: main |
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