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Cytoplasmic HuR Expression Enhances Chemoresistance in Pleural Mesothelioma Through Increased Expression of CALB2, Promotion of the E2F Pathway, and Suppression of the p53 Pathway.

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Title: Cytoplasmic HuR Expression Enhances Chemoresistance in Pleural Mesothelioma Through Increased Expression of CALB2, Promotion of the E2F Pathway, and Suppression of the p53 Pathway.
Authors: Kirimura, Susumu1 (AUTHOR) kirimura.pth2@tmd.ac.jp, Kurata, Morito2 (AUTHOR), Ishibashi, Hironori3 (AUTHOR), Taniguchi, Yusuke2 (AUTHOR), Kinowaki, Yuko2 (AUTHOR), Sugita, Keisuke2,4 (AUTHOR), Okubo, Kenichi3 (AUTHOR)
Source: Thoracic Cancer. Apr2025, Vol. 16 Issue 7, p1-9. 9p.
Database: Academic Search Ultimate
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  Data: Cytoplasmic HuR Expression Enhances Chemoresistance in Pleural Mesothelioma Through Increased Expression of CALB2, Promotion of the E2F Pathway, and Suppression of the p53 Pathway.
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  Data: <searchLink fieldCode="JN" term="%22Thoracic+Cancer%22">Thoracic Cancer</searchLink>. Apr2025, Vol. 16 Issue 7, p1-9. 9p.
PLink https://search.ebscohost.com/login.aspx?direct=true&site=eds-live&db=asn&AN=184466109
RecordInfo BibRecord:
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      – Type: doi
        Value: 10.1111/1759-7714.70062
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      – Code: eng
        Text: English
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        PageCount: 9
        StartPage: 1
    Titles:
      – TitleFull: Cytoplasmic HuR Expression Enhances Chemoresistance in Pleural Mesothelioma Through Increased Expression of CALB2, Promotion of the E2F Pathway, and Suppression of the p53 Pathway.
        Type: main
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          Name:
            NameFull: Kirimura, Susumu
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            NameFull: Kurata, Morito
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            NameFull: Ishibashi, Hironori
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            NameFull: Taniguchi, Yusuke
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            NameFull: Kinowaki, Yuko
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            NameFull: Sugita, Keisuke
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            NameFull: Okubo, Kenichi
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            – D: 01
              M: 04
              Text: Apr2025
              Type: published
              Y: 2025
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              Value: 16
            – Type: issue
              Value: 7
          Titles:
            – TitleFull: Thoracic Cancer
              Type: main
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