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Deacetylation of nuclear AIF provides a braking mechanism for caspase-independent chromatinolysis and necrotic brain injury.

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Title: Deacetylation of nuclear AIF provides a braking mechanism for caspase-independent chromatinolysis and necrotic brain injury.
Authors: Hu, Chen1 (AUTHOR), Geng, Jichuan1 (AUTHOR), Shan, Peipei2 (AUTHOR), Zhang, Tongqing1 (AUTHOR), Zhang, Zhuqing1 (AUTHOR), Zhang, Xiaoyu1 (AUTHOR), Lin, Menghan1 (AUTHOR), Zhang, Xiaoxia1 (AUTHOR), Chang, Dong1 (AUTHOR), He, Baokun3 (AUTHOR), Jia, Deshui4 (AUTHOR), Zhang, Mary5 (AUTHOR), Wang, Chuangui1 (AUTHOR) cgwang24@sdut.edu.cn, Zhang, Shengping1 (AUTHOR) spzhang@sdut.edu.cn
Source: Communications Biology. 5/27/2025, Vol. 8 Issue 1, p1-17. 17p.
Database: Academic Search Ultimate
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  Data: Deacetylation of nuclear AIF provides a braking mechanism for caspase-independent chromatinolysis and necrotic brain injury.
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  Data: <searchLink fieldCode="JN" term="%22Communications+Biology%22">Communications Biology</searchLink>. 5/27/2025, Vol. 8 Issue 1, p1-17. 17p.
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        Value: 10.1038/s42003-025-08255-w
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        Text: English
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      – TitleFull: Deacetylation of nuclear AIF provides a braking mechanism for caspase-independent chromatinolysis and necrotic brain injury.
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            – D: 27
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              Text: 5/27/2025
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              Y: 2025
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