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Homologous recombination-DNA damage response defects increase TMB and neoantigen load, but not effector T cell density and clonal diversity in pancreatic cancer.

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Title: Homologous recombination-DNA damage response defects increase TMB and neoantigen load, but not effector T cell density and clonal diversity in pancreatic cancer.
Authors: Lei, Mengyue1,2 (AUTHOR), Gai, Jessica3,4,5 (AUTHOR), McPhaul, Thomas J.3,5,6 (AUTHOR), Luo, Huijuan1,2 (AUTHOR), Lin, Penghui1,2 (AUTHOR), Liu, Dongbing1,2 (AUTHOR), Pishvaian, Michael3,4,5 (AUTHOR), Roberts, Nicholas J.3,4,7 (AUTHOR), Wu, Kui1,2 (AUTHOR), He, Jin3,4,5,6 (AUTHOR), Zheng, Lei3,4,5,6,8 (AUTHOR) zhengl@uthscsa.edu
Source: Experimental Hematology & Oncology. 6/18/2025, Vol. 14 Issue 1, p1-6. 6p.
Database: Academic Search Ultimate
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  Data: Homologous recombination-DNA damage response defects increase TMB and neoantigen load, but not effector T cell density and clonal diversity in pancreatic cancer.
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  Data: <searchLink fieldCode="JN" term="%22Experimental+Hematology+%26+Oncology%22">Experimental Hematology & Oncology</searchLink>. 6/18/2025, Vol. 14 Issue 1, p1-6. 6p.
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        Value: 10.1186/s40164-025-00673-0
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      – TitleFull: Homologous recombination-DNA damage response defects increase TMB and neoantigen load, but not effector T cell density and clonal diversity in pancreatic cancer.
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            – D: 18
              M: 06
              Text: 6/18/2025
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              Y: 2025
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