Homologous recombination-DNA damage response defects increase TMB and neoantigen load, but not effector T cell density and clonal diversity in pancreatic cancer.
Saved in:
| Title: | Homologous recombination-DNA damage response defects increase TMB and neoantigen load, but not effector T cell density and clonal diversity in pancreatic cancer. |
|---|---|
| Authors: | Lei, Mengyue1,2 (AUTHOR), Gai, Jessica3,4,5 (AUTHOR), McPhaul, Thomas J.3,5,6 (AUTHOR), Luo, Huijuan1,2 (AUTHOR), Lin, Penghui1,2 (AUTHOR), Liu, Dongbing1,2 (AUTHOR), Pishvaian, Michael3,4,5 (AUTHOR), Roberts, Nicholas J.3,4,7 (AUTHOR), Wu, Kui1,2 (AUTHOR), He, Jin3,4,5,6 (AUTHOR), Zheng, Lei3,4,5,6,8 (AUTHOR) zhengl@uthscsa.edu |
| Source: | Experimental Hematology & Oncology. 6/18/2025, Vol. 14 Issue 1, p1-6. 6p. |
| Database: | Academic Search Ultimate |
|
Full text is not displayed to guests.
Login for full access.
|
|
| ISSN: | 21623619 |
|---|---|
| DOI: | 10.1186/s40164-025-00673-0 |
