APA (7th ed.) Citation

De Luca, P., Mele, M., Tanqueiro, S., Napoli, F., Butkevičiūtė, U., Souto, A. C., . . . Duarte, C. B. (2025). Synaptic accumulation of GluN2B-containing NMDA receptors mediates the effects of BDNF-TrkB signalling on synaptic plasticity and in hyperexcitability during status epilepticus. Journal of Biomedical Science, 32(1), 1. https://doi.org/10.1186/s12929-025-01164-4

Chicago Style (17th ed.) Citation

De Luca, Pasqualino, et al. "Synaptic Accumulation of GluN2B-containing NMDA Receptors Mediates the Effects of BDNF-TrkB Signalling on Synaptic Plasticity and in Hyperexcitability During Status Epilepticus." Journal of Biomedical Science 32, no. 1 (2025): 1. https://doi.org/10.1186/s12929-025-01164-4.

MLA (9th ed.) Citation

De Luca, Pasqualino, et al. "Synaptic Accumulation of GluN2B-containing NMDA Receptors Mediates the Effects of BDNF-TrkB Signalling on Synaptic Plasticity and in Hyperexcitability During Status Epilepticus." Journal of Biomedical Science, vol. 32, no. 1, 2025, p. 1, https://doi.org/10.1186/s12929-025-01164-4.