Synaptic accumulation of GluN2B-containing NMDA receptors mediates the effects of BDNF-TrkB signalling on synaptic plasticity and in hyperexcitability during status epilepticus.
Saved in:
| Title: | Synaptic accumulation of GluN2B-containing NMDA receptors mediates the effects of BDNF-TrkB signalling on synaptic plasticity and in hyperexcitability during status epilepticus. |
|---|---|
| Authors: | De Luca, Pasqualino1,2,3 (AUTHOR), Mele, Miranda1,2 (AUTHOR), Tanqueiro, Sara4 (AUTHOR), Napoli, Francesca1 (AUTHOR), Butkevičiūtė, Ugné1 (AUTHOR), Souto, Arthur C.1 (AUTHOR), Costa, Rui O.1,2 (AUTHOR), Schwarz, Alexander1,5 (AUTHOR), Drexel, Meinrad6 (AUTHOR), Sebastião, Ana M.4 (AUTHOR), Diógenes, Maria J.4 (AUTHOR), Duarte, Carlos B.1,7 (AUTHOR) cbduarte@ci.uc.pt |
| Source: | Journal of Biomedical Science. 9/1/2025, Vol. 32 Issue 1, p1-18. 18p. |
| Database: | Academic Search Ultimate |
|
Full text is not displayed to guests.
Login for full access.
|
|
| ISSN: | 10217770 |
|---|---|
| DOI: | 10.1186/s12929-025-01164-4 |