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Targeting METTL16 attenuates mesangial cell viability and fibrosis in a high-glucose state by suppressing m6A modification and the expression of RAP1B.

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Title: Targeting METTL16 attenuates mesangial cell viability and fibrosis in a high-glucose state by suppressing m6A modification and the expression of RAP1B.
Authors: Wang, Weixu1 (AUTHOR) wentang86995322093@163.com, Luo, Yanhong1 (AUTHOR)
Source: BMC Molecular & Cell Biology. 10/28/2025, Vol. 26 Issue 1, p1-10. 10p.
Database: Academic Search Ultimate
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PubType: Academic Journal
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  Data: Targeting METTL16 attenuates mesangial cell viability and fibrosis in a high-glucose state by suppressing m6A modification and the expression of RAP1B.
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  Data: <searchLink fieldCode="AR" term="%22Wang%2C+Weixu%22">Wang, Weixu</searchLink><relatesTo>1</relatesTo> (AUTHOR)<i> wentang86995322093@163.com</i><br /><searchLink fieldCode="AR" term="%22Luo%2C+Yanhong%22">Luo, Yanhong</searchLink><relatesTo>1</relatesTo> (AUTHOR)
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  Data: <searchLink fieldCode="JN" term="%22BMC+Molecular+%26+Cell+Biology%22">BMC Molecular & Cell Biology</searchLink>. 10/28/2025, Vol. 26 Issue 1, p1-10. 10p.
PLink https://search.ebscohost.com/login.aspx?direct=true&site=eds-live&db=asn&AN=188952487
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        Value: 10.1186/s12860-025-00559-3
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      – Code: eng
        Text: English
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      – TitleFull: Targeting METTL16 attenuates mesangial cell viability and fibrosis in a high-glucose state by suppressing m6A modification and the expression of RAP1B.
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              Text: 10/28/2025
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