Dysregulated lactate metabolism synergizes with ALS genetic risk factors to accelerate motor decline.
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| Title: | Dysregulated lactate metabolism synergizes with ALS genetic risk factors to accelerate motor decline. |
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| Authors: | Tendulkar, Shweta1 (AUTHOR), Wu, Tong2 (AUTHOR), Strickland, Amy2 (AUTHOR), Hackett, Amber R.2 (AUTHOR), Sato-Yamada, Yurie3 (AUTHOR), Mao, Xianrong2 (AUTHOR), Sasaki, Yo2 (AUTHOR), Milbrandt, Jeffrey2,4 (AUTHOR), Bloom, A. Joseph2,4 (AUTHOR) ajbloom@wustl.edu, DiAntonio, Aaron1,4 (AUTHOR) diantonio@wustl.edu |
| Source: | PLoS ONE. 4/17/2026, Vol. 21 Issue 4, p1-15. 15p. |
| Database: | Academic Search Ultimate |
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| FullText | Links: – Type: pdflink Text: Availability: 1 |
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| Header | DbId: asn DbLabel: Academic Search Ultimate An: 193088262 AccessLevel: 2 PubType: Academic Journal PubTypeId: academicJournal PreciseRelevancyScore: 0 |
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| PLink | https://search.ebscohost.com/login.aspx?direct=true&site=eds-live&db=asn&AN=193088262 |
| RecordInfo | BibRecord: BibEntity: Identifiers: – Type: doi Value: 10.1371/journal.pone.0347135 Languages: – Code: eng Text: English PhysicalDescription: Pagination: PageCount: 15 StartPage: 1 Titles: – TitleFull: Dysregulated lactate metabolism synergizes with ALS genetic risk factors to accelerate motor decline. Type: main BibRelationships: HasContributorRelationships: – PersonEntity: Name: NameFull: Tendulkar, Shweta – PersonEntity: Name: NameFull: Wu, Tong – PersonEntity: Name: NameFull: Strickland, Amy – PersonEntity: Name: NameFull: Hackett, Amber R. – PersonEntity: Name: NameFull: Sato-Yamada, Yurie – PersonEntity: Name: NameFull: Mao, Xianrong – PersonEntity: Name: NameFull: Sasaki, Yo – PersonEntity: Name: NameFull: Milbrandt, Jeffrey – PersonEntity: Name: NameFull: Bloom, A. Joseph – PersonEntity: Name: NameFull: DiAntonio, Aaron IsPartOfRelationships: – BibEntity: Dates: – D: 17 M: 04 Text: 4/17/2026 Type: published Y: 2026 Identifiers: – Type: issn-print Value: 19326203 Numbering: – Type: volume Value: 21 – Type: issue Value: 4 Titles: – TitleFull: PLoS ONE Type: main |
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