In Vivo ORF Overexpression Screening Identifies CCN4 as a Regulator of Glioblastoma Growth Validated Across Multiple Models.
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| Title: | In Vivo ORF Overexpression Screening Identifies CCN4 as a Regulator of Glioblastoma Growth Validated Across Multiple Models. |
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| Authors: | Dasgupta, Pushan1,2,3,4 (AUTHOR) |
| Source: | International Journal of Molecular Sciences. Jun2026, Vol. 27 Issue 12, p5227. 18p. |
| Database: | Academic Search Ultimate |
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| FullText | Links: – Type: pdflink Text: Availability: 1 |
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| Header | DbId: asn DbLabel: Academic Search Ultimate An: 194911465 AccessLevel: 2 PubType: Academic Journal PubTypeId: academicJournal PreciseRelevancyScore: 0 |
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| Items | – Name: Title Label: Title Group: Ti Data: In Vivo ORF Overexpression Screening Identifies CCN4 as a Regulator of Glioblastoma Growth Validated Across Multiple Models. – Name: Author Label: Authors Group: Au Data: <searchLink fieldCode="AR" term="%22Dasgupta%2C+Pushan%22">Dasgupta, Pushan</searchLink><relatesTo>1,2,3,4</relatesTo> (AUTHOR) – Name: TitleSource Label: Source Group: Src Data: <searchLink fieldCode="JN" term="%22International+Journal+of+Molecular+Sciences%22">International Journal of Molecular Sciences</searchLink>. Jun2026, Vol. 27 Issue 12, p5227. 18p. |
| PLink | https://search.ebscohost.com/login.aspx?direct=true&site=eds-live&db=asn&AN=194911465 |
| RecordInfo | BibRecord: BibEntity: Identifiers: – Type: doi Value: 10.3390/ijms27125227 Languages: – Code: eng Text: English PhysicalDescription: Pagination: PageCount: 18 StartPage: 5227 Titles: – TitleFull: In Vivo ORF Overexpression Screening Identifies CCN4 as a Regulator of Glioblastoma Growth Validated Across Multiple Models. Type: main BibRelationships: HasContributorRelationships: – PersonEntity: Name: NameFull: Dasgupta, Pushan IsPartOfRelationships: – BibEntity: Dates: – D: 15 M: 06 Text: Jun2026 Type: published Y: 2026 Identifiers: – Type: issn-print Value: 16616596 Numbering: – Type: volume Value: 27 – Type: issue Value: 12 Titles: – TitleFull: International Journal of Molecular Sciences Type: main |
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