Matveyenko, A. V., & Butler, P. C. (2006). β-Cell Deficit Due to Increased Apoptosis in the Human Islet Amyloid Polypeptide Transgenic (HIP) Rat Recapitulates the Metabolic Defects Present in Type 2 Diabetes. Diabetes, 55(7), 2106. https://doi.org/10.2337/db05-1672
Chicago Style (17th ed.) CitationMatveyenko, Aleksey V., and Peter C. Butler. "β-Cell Deficit Due to Increased Apoptosis in the Human Islet Amyloid Polypeptide Transgenic (HIP) Rat Recapitulates the Metabolic Defects Present in Type 2 Diabetes." Diabetes 55, no. 7 (2006): 2106. https://doi.org/10.2337/db05-1672.
MLA (9th ed.) CitationMatveyenko, Aleksey V., and Peter C. Butler. "β-Cell Deficit Due to Increased Apoptosis in the Human Islet Amyloid Polypeptide Transgenic (HIP) Rat Recapitulates the Metabolic Defects Present in Type 2 Diabetes." Diabetes, vol. 55, no. 7, 2006, p. 2106, https://doi.org/10.2337/db05-1672.