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β-Cell Deficit Due to Increased Apoptosis in the Human Islet Amyloid Polypeptide Transgenic (HIP) Rat Recapitulates the Metabolic Defects Present in Type 2 Diabetes.

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Title: β-Cell Deficit Due to Increased Apoptosis in the Human Islet Amyloid Polypeptide Transgenic (HIP) Rat Recapitulates the Metabolic Defects Present in Type 2 Diabetes.
Authors: Matveyenko, Aleksey V.1, Butler, Peter C.1 pbutler@mednet.ucla.edu
Source: Diabetes. Jul2006, Vol. 55 Issue 7, p2106-2114. 9p. 2 Charts, 6 Graphs.
Database: Academic Search Ultimate
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  Data: β-Cell Deficit Due to Increased Apoptosis in the Human Islet Amyloid Polypeptide Transgenic (HIP) Rat Recapitulates the Metabolic Defects Present in Type 2 Diabetes.
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  Data: <searchLink fieldCode="JN" term="%22Diabetes%22">Diabetes</searchLink>. Jul2006, Vol. 55 Issue 7, p2106-2114. 9p. 2 Charts, 6 Graphs.
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      – Type: doi
        Value: 10.2337/db05-1672
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      – Code: eng
        Text: English
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        PageCount: 9
        StartPage: 2106
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      – TitleFull: β-Cell Deficit Due to Increased Apoptosis in the Human Islet Amyloid Polypeptide Transgenic (HIP) Rat Recapitulates the Metabolic Defects Present in Type 2 Diabetes.
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            NameFull: Matveyenko, Aleksey V.
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            NameFull: Butler, Peter C.
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            – D: 01
              M: 07
              Text: Jul2006
              Type: published
              Y: 2006
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            – TitleFull: Diabetes
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