APA (7th ed.) Citation

Kreuter, A., Wieland, U., Gambichler, T., Altmeyer, P., Pfister, H., Tenner-Racz, K., . . . Brockmeyer, N. (2007). p16ink4a expression decreases during imiquimod treatment of anal intraepithelial neoplasia in human immunodeficiency virus-infected men and correlates with the decline of lesional high-risk human papillomavirus DNA load. British Journal of Dermatology, 157(3), 523. https://doi.org/10.1111/j.1365-2133.2007.08004.x

Chicago Style (17th ed.) Citation

Kreuter, A., U. Wieland, T. Gambichler, P. Altmeyer, H. Pfister, K. Tenner-Racz, P. Racz, A. Potthoff, and N.H Brockmeyer. "P16ink4a Expression Decreases During Imiquimod Treatment of Anal Intraepithelial Neoplasia in Human Immunodeficiency Virus-infected Men and Correlates with the Decline of Lesional High-risk Human Papillomavirus DNA Load." British Journal of Dermatology 157, no. 3 (2007): 523. https://doi.org/10.1111/j.1365-2133.2007.08004.x.

MLA (9th ed.) Citation

Kreuter, A., et al. "P16ink4a Expression Decreases During Imiquimod Treatment of Anal Intraepithelial Neoplasia in Human Immunodeficiency Virus-infected Men and Correlates with the Decline of Lesional High-risk Human Papillomavirus DNA Load." British Journal of Dermatology, vol. 157, no. 3, 2007, p. 523, https://doi.org/10.1111/j.1365-2133.2007.08004.x.