Mutations of the same conserved glutamate residue in NBD2 of the SUR1 subunit of the KATP channel can result in either hyperinsulinism or neonatal diabetes.
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| Title: | Mutations of the same conserved glutamate residue in NBD2 of the SUR1 subunit of the K |
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| Authors: | Mannikko, R.1, Flanagan, S. E.2, Sim, X.1, Segal, D.3, Hussain, K.4, Ellard, S.2, Hattersley, A. T.2, Ashcroft, F. M.1 |
| Source: | Proceedings of the Physiological Society. 2011, Vol. 23, p31P-31P. 1/2p. |
| Database: | Academic Search Ultimate |
| FullText | Links: – Type: pdflink Text: Availability: 0 |
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| Header | DbId: asn DbLabel: Academic Search Ultimate An: 67429973 AccessLevel: 2 PubType: Conference PubTypeId: conference PreciseRelevancyScore: 0 |
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| Items | – Name: Title Label: Title Group: Ti Data: Mutations of the same conserved glutamate residue in NBD2 of the SUR1 subunit of the K<subscript>ATP</subscript> channel can result in either hyperinsulinism or neonatal diabetes. – Name: Author Label: Authors Group: Au Data: <searchLink fieldCode="AR" term="%22Mannikko%2C+R%2E%22">Mannikko, R.</searchLink><relatesTo>1</relatesTo><br /><searchLink fieldCode="AR" term="%22Flanagan%2C+S%2E+E%2E%22">Flanagan, S. E.</searchLink><relatesTo>2</relatesTo><br /><searchLink fieldCode="AR" term="%22Sim%2C+X%2E%22">Sim, X.</searchLink><relatesTo>1</relatesTo><br /><searchLink fieldCode="AR" term="%22Segal%2C+D%2E%22">Segal, D.</searchLink><relatesTo>3</relatesTo><br /><searchLink fieldCode="AR" term="%22Hussain%2C+K%2E%22">Hussain, K.</searchLink><relatesTo>4</relatesTo><br /><searchLink fieldCode="AR" term="%22Ellard%2C+S%2E%22">Ellard, S.</searchLink><relatesTo>2</relatesTo><br /><searchLink fieldCode="AR" term="%22Hattersley%2C+A%2E+T%2E%22">Hattersley, A. T.</searchLink><relatesTo>2</relatesTo><br /><searchLink fieldCode="AR" term="%22Ashcroft%2C+F%2E+M%2E%22">Ashcroft, F. M.</searchLink><relatesTo>1</relatesTo> – Name: TitleSource Label: Source Group: Src Data: <searchLink fieldCode="JN" term="%22Proceedings+of+the+Physiological+Society%22">Proceedings of the Physiological Society</searchLink>. 2011, Vol. 23, p31P-31P. 1/2p. |
| PLink | https://search.ebscohost.com/login.aspx?direct=true&site=eds-live&db=asn&AN=67429973 |
| RecordInfo | BibRecord: BibEntity: Languages: – Code: eng Text: English PhysicalDescription: Pagination: PageCount: 0 StartPage: 31P Titles: – TitleFull: Mutations of the same conserved glutamate residue in NBD2 of the SUR1 subunit of the KATP channel can result in either hyperinsulinism or neonatal diabetes. Type: main BibRelationships: HasContributorRelationships: – PersonEntity: Name: NameFull: Mannikko, R. – PersonEntity: Name: NameFull: Flanagan, S. E. – PersonEntity: Name: NameFull: Sim, X. – PersonEntity: Name: NameFull: Segal, D. – PersonEntity: Name: NameFull: Hussain, K. – PersonEntity: Name: NameFull: Ellard, S. – PersonEntity: Name: NameFull: Hattersley, A. T. – PersonEntity: Name: NameFull: Ashcroft, F. M. IsPartOfRelationships: – BibEntity: Dates: – D: 01 M: 06 Text: 2011 Type: published Y: 2011 Identifiers: – Type: issn-print Value: 17496187 Numbering: – Type: volume Value: 23 Titles: – TitleFull: Proceedings of the Physiological Society Type: main |
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