GS, S., JI, L., T, S., N, H., AM, K., PJ, B., & AM, T. (2001). Residual ataxia telangiectasia mutated protein function in cells from ataxia telangiectasia patients, with 5762ins137 and 7271T-->G mutations, showing a less severe phenotype. The Journal of biological chemistry, 276(32), 30133. https://doi.org/10.1074/jbc.M103160200
Chicago Style (17th ed.) CitationGS, Stewart, Last JI, Stankovic T, Haites N, Kidd AM, Byrd PJ, and Taylor AM. "Residual Ataxia Telangiectasia Mutated Protein Function in Cells from Ataxia Telangiectasia Patients, with 5762ins137 and 7271T-->G Mutations, Showing a Less Severe Phenotype." The Journal of Biological Chemistry 276, no. 32 (2001): 30133. https://doi.org/10.1074/jbc.M103160200.
MLA (9th ed.) CitationGS, Stewart, et al. "Residual Ataxia Telangiectasia Mutated Protein Function in Cells from Ataxia Telangiectasia Patients, with 5762ins137 and 7271T-->G Mutations, Showing a Less Severe Phenotype." The Journal of Biological Chemistry, vol. 276, no. 32, 2001, p. 30133, https://doi.org/10.1074/jbc.M103160200.