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Residual ataxia telangiectasia mutated protein function in cells from ataxia telangiectasia patients, with 5762ins137 and 7271T-->G mutations, showing a less severe phenotype.

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Title: Residual ataxia telangiectasia mutated protein function in cells from ataxia telangiectasia patients, with 5762ins137 and 7271T-->G mutations, showing a less severe phenotype.
Authors: Stewart GS; CRC Institute for Cancer Studies, the University of Birmingham, Vincent Drive, Edgbaston, Birmingham B15 2TT, United Kingdom., Last JI, Stankovic T, Haites N, Kidd AM, Byrd PJ, Taylor AM
Source: The Journal of biological chemistry [J Biol Chem] 2001 Aug 10; Vol. 276 (32), pp. 30133-41. Date of Electronic Publication: 2001 May 29.
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
Journal Info: Publisher: Elsevier Inc. on behalf of American Society for Biochemistry and Molecular Biology Country of Publication: United States NLM ID: 2985121R Publication Model: Print-Electronic Cited Medium: Print ISSN: 0021-9258 (Print) Linking ISSN: 00219258 NLM ISO Abbreviation: J Biol Chem Subsets: MEDLINE
Database: MEDLINE Ultimate
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DbLabel: MEDLINE Ultimate
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AccessLevel: 2
PubType: Academic Journal
PubTypeId: academicJournal
PreciseRelevancyScore: 0
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  Data: Residual ataxia telangiectasia mutated protein function in cells from ataxia telangiectasia patients, with 5762ins137 and 7271T-->G mutations, showing a less severe phenotype.
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  Data: <searchLink fieldCode="AU" term="%22Stewart+GS%22">Stewart GS</searchLink>; CRC Institute for Cancer Studies, the University of Birmingham, Vincent Drive, Edgbaston, Birmingham B15 2TT, United Kingdom.<br /><searchLink fieldCode="AU" term="%22Last+JI%22">Last JI</searchLink><br /><searchLink fieldCode="AU" term="%22Stankovic+T%22">Stankovic T</searchLink><br /><searchLink fieldCode="AU" term="%22Haites+N%22">Haites N</searchLink><br /><searchLink fieldCode="AU" term="%22Kidd+AM%22">Kidd AM</searchLink><br /><searchLink fieldCode="AU" term="%22Byrd+PJ%22">Byrd PJ</searchLink><br /><searchLink fieldCode="AU" term="%22Taylor+AM%22">Taylor AM</searchLink>
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  Data: <searchLink fieldCode="JN" term="%222985121R%22">The Journal of biological chemistry</searchLink> [J Biol Chem] 2001 Aug 10; Vol. 276 (32), pp. 30133-41. <i>Date of Electronic Publication: </i>2001 May 29.
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  Data: Journal Article; Research Support, Non-U.S. Gov't
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  Data: <i>Publisher: </i><searchLink fieldCode="PB" term="%22Elsevier+Inc%2E+on+behalf+of+American+Society+for+Biochemistry+and+Molecular+Biology%22">Elsevier Inc. on behalf of American Society for Biochemistry and Molecular Biology </searchLink><i>Country of Publication: </i>United States <i>NLM ID: </i>2985121R <i>Publication Model: </i>Print-Electronic <i>Cited Medium: </i>Print <i>ISSN: </i>0021-9258 (Print) <i>Linking ISSN: </i><searchLink fieldCode="IS" term="%2200219258%22">00219258 </searchLink><i>NLM ISO Abbreviation: </i>J Biol Chem <i>Subsets: </i>MEDLINE
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      – Type: doi
        Value: 10.1074/jbc.M103160200
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      – Code: eng
        Text: English
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        StartPage: 30133
    Titles:
      – TitleFull: Residual ataxia telangiectasia mutated protein function in cells from ataxia telangiectasia patients, with 5762ins137 and 7271T-->G mutations, showing a less severe phenotype.
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            NameFull: Stewart GS
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            NameFull: Last JI
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            NameFull: Stankovic T
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            NameFull: Haites N
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            NameFull: Kidd AM
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            NameFull: Byrd PJ
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            – D: 10
              M: 08
              Text: 2001 Aug 10
              Type: published
              Y: 2001
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              Value: 0021-9258
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              Value: 276
            – Type: issue
              Value: 32
          Titles:
            – TitleFull: The Journal of biological chemistry
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