Calcitonin gene-related peptide is not essential for the development of pressure overload-induced hypertrophy in vivo.
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| Title: | Calcitonin gene-related peptide is not essential for the development of pressure overload-induced hypertrophy in vivo. |
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| Authors: | Young ME; Department of Internal Medicine, University of Texas-Houston Medical School, 77030, USA., Okerberg KA, Wilson CR, Deferrari DA, Ying J, Guthrie P, Razeghi P, Clubb FJ Jr, Taegtmeyer H |
| Source: | Molecular and cellular biochemistry [Mol Cell Biochem] 2001 Sep; Vol. 225 (1-), pp. 43-9. |
| Publication Type: | Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S. |
| Journal Info: | Publisher: Springer Country of Publication: Netherlands NLM ID: 0364456 Publication Model: Print Cited Medium: Print ISSN: 0300-8177 (Print) Linking ISSN: 03008177 NLM ISO Abbreviation: Mol Cell Biochem Subsets: MEDLINE |
| Database: | MEDLINE Ultimate |
| FullText | Text: Availability: 0 |
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| Header | DbId: mdl DbLabel: MEDLINE Ultimate An: 11716363 AccessLevel: 2 PubType: Academic Journal PubTypeId: academicJournal PreciseRelevancyScore: 0 |
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| Items | – Name: Title Label: Title Group: Ti Data: Calcitonin gene-related peptide is not essential for the development of pressure overload-induced hypertrophy in vivo. – Name: Author Label: Authors Group: Au Data: <searchLink fieldCode="AU" term="%22Young+ME%22">Young ME</searchLink>; Department of Internal Medicine, University of Texas-Houston Medical School, 77030, USA.<br /><searchLink fieldCode="AU" term="%22Okerberg+KA%22">Okerberg KA</searchLink><br /><searchLink fieldCode="AU" term="%22Wilson+CR%22">Wilson CR</searchLink><br /><searchLink fieldCode="AU" term="%22Deferrari+DA%22">Deferrari DA</searchLink><br /><searchLink fieldCode="AU" term="%22Ying+J%22">Ying J</searchLink><br /><searchLink fieldCode="AU" term="%22Guthrie+P%22">Guthrie P</searchLink><br /><searchLink fieldCode="AU" term="%22Razeghi+P%22">Razeghi P</searchLink><br /><searchLink fieldCode="AU" term="%22Clubb+FJ+Jr%22">Clubb FJ Jr</searchLink><br /><searchLink fieldCode="AU" term="%22Taegtmeyer+H%22">Taegtmeyer H</searchLink> – Name: TitleSource Label: Source Group: Src Data: <searchLink fieldCode="JN" term="%220364456%22">Molecular and cellular biochemistry</searchLink> [Mol Cell Biochem] 2001 Sep; Vol. 225 (1-), pp. 43-9. – Name: TypePub Label: Publication Type Group: TypPub Data: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S. – Name: TitleSource Label: Journal Info Group: Src Data: <i>Publisher: </i><searchLink fieldCode="PB" term="%22Springer%22">Springer </searchLink><i>Country of Publication: </i>Netherlands <i>NLM ID: </i>0364456 <i>Publication Model: </i>Print <i>Cited Medium: </i>Print <i>ISSN: </i>0300-8177 (Print) <i>Linking ISSN: </i><searchLink fieldCode="IS" term="%2203008177%22">03008177 </searchLink><i>NLM ISO Abbreviation: </i>Mol Cell Biochem <i>Subsets: </i>MEDLINE |
| PLink | https://search.ebscohost.com/login.aspx?direct=true&site=eds-live&db=mdl&AN=11716363 |
| RecordInfo | BibRecord: BibEntity: Identifiers: – Type: doi Value: 10.1023/a:1012212722749 Languages: – Code: eng Text: English PhysicalDescription: Pagination: StartPage: 43 Titles: – TitleFull: Calcitonin gene-related peptide is not essential for the development of pressure overload-induced hypertrophy in vivo. Type: main BibRelationships: HasContributorRelationships: – PersonEntity: Name: NameFull: Young ME – PersonEntity: Name: NameFull: Okerberg KA – PersonEntity: Name: NameFull: Wilson CR – PersonEntity: Name: NameFull: Deferrari DA – PersonEntity: Name: NameFull: Ying J – PersonEntity: Name: NameFull: Guthrie P – PersonEntity: Name: NameFull: Razeghi P – PersonEntity: Name: NameFull: Clubb FJ Jr – PersonEntity: Name: NameFull: Taegtmeyer H IsPartOfRelationships: – BibEntity: Dates: – D: 01 M: 09 Text: 2001 Sep Type: published Y: 2001 Identifiers: – Type: issn-print Value: 0300-8177 Numbering: – Type: volume Value: 225 – Type: issue Value: 1- Titles: – TitleFull: Molecular and cellular biochemistry Type: main |
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