Histone deacetylase inhibitors decrease Toll-like receptor-mediated activation of proinflammatory gene expression by impairing transcription factor recruitment.
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| Title: | Histone deacetylase inhibitors decrease Toll-like receptor-mediated activation of proinflammatory gene expression by impairing transcription factor recruitment. |
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| Authors: | Bode KA; Department of Medical Microbiology and Hygiene, University Heidelberg, Heidelberg, Germany., Schroder K, Hume DA, Ravasi T, Heeg K, Sweet MJ, Dalpke AH |
| Source: | Immunology [Immunology] 2007 Dec; Vol. 122 (4), pp. 596-606. Date of Electronic Publication: 2007 Jul 16. |
| Publication Type: | Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't |
| Journal Info: | Publisher: Blackwell Scientific Publications Country of Publication: England NLM ID: 0374672 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1365-2567 (Electronic) Linking ISSN: 00192805 NLM ISO Abbreviation: Immunology Subsets: MEDLINE |
| Database: | MEDLINE Ultimate |
| FullText | Links: – Type: pdflink Text: Availability: 0 |
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| Header | DbId: mdl DbLabel: MEDLINE Ultimate An: 17635610 AccessLevel: 2 PubType: Academic Journal PubTypeId: academicJournal PreciseRelevancyScore: 0 |
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| Items | – Name: Title Label: Title Group: Ti Data: Histone deacetylase inhibitors decrease Toll-like receptor-mediated activation of proinflammatory gene expression by impairing transcription factor recruitment. – Name: Author Label: Authors Group: Au Data: <searchLink fieldCode="AU" term="%22Bode+KA%22">Bode KA</searchLink>; Department of Medical Microbiology and Hygiene, University Heidelberg, Heidelberg, Germany.<br /><searchLink fieldCode="AU" term="%22Schroder+K%22">Schroder K</searchLink><br /><searchLink fieldCode="AU" term="%22Hume+DA%22">Hume DA</searchLink><br /><searchLink fieldCode="AU" term="%22Ravasi+T%22">Ravasi T</searchLink><br /><searchLink fieldCode="AU" term="%22Heeg+K%22">Heeg K</searchLink><br /><searchLink fieldCode="AU" term="%22Sweet+MJ%22">Sweet MJ</searchLink><br /><searchLink fieldCode="AU" term="%22Dalpke+AH%22">Dalpke AH</searchLink> – Name: TitleSource Label: Source Group: Src Data: <searchLink fieldCode="JN" term="%220374672%22">Immunology</searchLink> [Immunology] 2007 Dec; Vol. 122 (4), pp. 596-606. <i>Date of Electronic Publication: </i>2007 Jul 16. – Name: TypePub Label: Publication Type Group: TypPub Data: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't – Name: TitleSource Label: Journal Info Group: Src Data: <i>Publisher: </i><searchLink fieldCode="PB" term="%22Blackwell+Scientific+Publications%22">Blackwell Scientific Publications </searchLink><i>Country of Publication: </i>England <i>NLM ID: </i>0374672 <i>Publication Model: </i>Print-Electronic <i>Cited Medium: </i>Internet <i>ISSN: </i>1365-2567 (Electronic) <i>Linking ISSN: </i><searchLink fieldCode="IS" term="%2200192805%22">00192805 </searchLink><i>NLM ISO Abbreviation: </i>Immunology <i>Subsets: </i>MEDLINE |
| PLink | https://search.ebscohost.com/login.aspx?direct=true&site=eds-live&db=mdl&AN=17635610 |
| RecordInfo | BibRecord: BibEntity: Identifiers: – Type: doi Value: 10.1111/j.1365-2567.2007.02678.x Languages: – Code: eng Text: English PhysicalDescription: Pagination: StartPage: 596 Titles: – TitleFull: Histone deacetylase inhibitors decrease Toll-like receptor-mediated activation of proinflammatory gene expression by impairing transcription factor recruitment. Type: main BibRelationships: HasContributorRelationships: – PersonEntity: Name: NameFull: Bode KA – PersonEntity: Name: NameFull: Schroder K – PersonEntity: Name: NameFull: Hume DA – PersonEntity: Name: NameFull: Ravasi T – PersonEntity: Name: NameFull: Heeg K – PersonEntity: Name: NameFull: Sweet MJ – PersonEntity: Name: NameFull: Dalpke AH IsPartOfRelationships: – BibEntity: Dates: – D: 01 M: 12 Text: 2007 Dec Type: published Y: 2007 Identifiers: – Type: issn-electronic Value: 1365-2567 Numbering: – Type: volume Value: 122 – Type: issue Value: 4 Titles: – TitleFull: Immunology Type: main |
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