APA (7th ed.) Citation

CF, L., AL, A., SW, L., KF, N., M, F., YW, K., . . . PM, V. (2010). Endothelium-derived nitric oxide inhibits the relaxation of the porcine coronary artery to natriuretic peptides by desensitizing big conductance calcium-activated potassium channels of vascular smooth muscle. The Journal of pharmacology and experimental therapeutics, 334(1), 223. https://doi.org/10.1124/jpet.110.166652

Chicago Style (17th ed.) Citation

CF, Liang, Au AL, Leung SW, Ng KF, Félétou M, Kwan YW, Man RY, and Vanhoutte PM. "Endothelium-derived Nitric Oxide Inhibits the Relaxation of the Porcine Coronary Artery to Natriuretic Peptides by Desensitizing Big Conductance Calcium-activated Potassium Channels of Vascular Smooth Muscle." The Journal of Pharmacology and Experimental Therapeutics 334, no. 1 (2010): 223. https://doi.org/10.1124/jpet.110.166652.

MLA (9th ed.) Citation

CF, Liang, et al. "Endothelium-derived Nitric Oxide Inhibits the Relaxation of the Porcine Coronary Artery to Natriuretic Peptides by Desensitizing Big Conductance Calcium-activated Potassium Channels of Vascular Smooth Muscle." The Journal of Pharmacology and Experimental Therapeutics, vol. 334, no. 1, 2010, p. 223, https://doi.org/10.1124/jpet.110.166652.