APA (7th ed.) Citation

DR, P., EP, J., NR, A., JD, D., & IF, P. (2013). Bortezomib-induced sensitization of malignant human glioma cells to vorinostat-induced apoptosis depends on reactive oxygen species production, mitochondrial dysfunction, Noxa upregulation, Mcl-1 cleavage, and DNA damage. Molecular carcinogenesis, 52(2), 118. https://doi.org/10.1002/mc.21835

Chicago Style (17th ed.) Citation

DR, Premkumar, Jane EP, Agostino NR, DiDomenico JD, and Pollack IF. "Bortezomib-induced Sensitization of Malignant Human Glioma Cells to Vorinostat-induced Apoptosis Depends on Reactive Oxygen Species Production, Mitochondrial Dysfunction, Noxa Upregulation, Mcl-1 Cleavage, and DNA Damage." Molecular Carcinogenesis 52, no. 2 (2013): 118. https://doi.org/10.1002/mc.21835.

MLA (9th ed.) Citation

DR, Premkumar, et al. "Bortezomib-induced Sensitization of Malignant Human Glioma Cells to Vorinostat-induced Apoptosis Depends on Reactive Oxygen Species Production, Mitochondrial Dysfunction, Noxa Upregulation, Mcl-1 Cleavage, and DNA Damage." Molecular Carcinogenesis, vol. 52, no. 2, 2013, p. 118, https://doi.org/10.1002/mc.21835.