Bortezomib-induced sensitization of malignant human glioma cells to vorinostat-induced apoptosis depends on reactive oxygen species production, mitochondrial dysfunction, Noxa upregulation, Mcl-1 cleavage, and DNA damage.

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Bibliographic Details
Title: Bortezomib-induced sensitization of malignant human glioma cells to vorinostat-induced apoptosis depends on reactive oxygen species production, mitochondrial dysfunction, Noxa upregulation, Mcl-1 cleavage, and DNA damage.
Authors: Premkumar DR; Department of Neurosurgery, Children's Hospital of Pittsburgh, Pittsburgh, Pennsylvania 15223, USA., Jane EP, Agostino NR, DiDomenico JD, Pollack IF
Source: Molecular carcinogenesis [Mol Carcinog] 2013 Feb; Vol. 52 (2), pp. 118-33. Date of Electronic Publication: 2011 Nov 15.
Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
Journal Info: Publisher: Wiley-Liss Country of Publication: United States NLM ID: 8811105 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1098-2744 (Electronic) Linking ISSN: 08991987 NLM ISO Abbreviation: Mol Carcinog Subsets: MEDLINE
Database: MEDLINE Ultimate
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