Exendin-4 attenuates high glucose-induced cardiomyocyte apoptosis via inhibition of endoplasmic reticulum stress and activation of SERCA2a.
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| Title: | Exendin-4 attenuates high glucose-induced cardiomyocyte apoptosis via inhibition of endoplasmic reticulum stress and activation of SERCA2a. |
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| Authors: | Younce CW; Metabolic Signaling and Disease Program, Diabetes and Obesity Research Center, Sanford Burnham Medical Research Institute at Lake Nona, 6400 Sanger Rd., Orlando, FL 32837, USA., Burmeister MA, Ayala JE |
| Source: | American journal of physiology. Cell physiology [Am J Physiol Cell Physiol] 2013 Mar; Vol. 304 (6), pp. C508-18. Date of Electronic Publication: 2013 Jan 09. |
| Publication Type: | Journal Article; Research Support, Non-U.S. Gov't |
| Journal Info: | Publisher: American Physiological Society Country of Publication: United States NLM ID: 100901225 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1522-1563 (Electronic) Linking ISSN: 03636143 NLM ISO Abbreviation: Am J Physiol Cell Physiol Subsets: MEDLINE |
| Database: | MEDLINE Ultimate |
| FullText | Links: – Type: pdflink Text: Availability: 0 |
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| Header | DbId: mdl DbLabel: MEDLINE Ultimate An: 23302777 AccessLevel: 2 PubType: Academic Journal PubTypeId: academicJournal PreciseRelevancyScore: 0 |
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| Items | – Name: Title Label: Title Group: Ti Data: Exendin-4 attenuates high glucose-induced cardiomyocyte apoptosis via inhibition of endoplasmic reticulum stress and activation of SERCA2a. – Name: Author Label: Authors Group: Au Data: <searchLink fieldCode="AU" term="%22Younce+CW%22">Younce CW</searchLink>; Metabolic Signaling and Disease Program, Diabetes and Obesity Research Center, Sanford Burnham Medical Research Institute at Lake Nona, 6400 Sanger Rd., Orlando, FL 32837, USA.<br /><searchLink fieldCode="AU" term="%22Burmeister+MA%22">Burmeister MA</searchLink><br /><searchLink fieldCode="AU" term="%22Ayala+JE%22">Ayala JE</searchLink> – Name: TitleSource Label: Source Group: Src Data: <searchLink fieldCode="JN" term="%22100901225%22">American journal of physiology. Cell physiology</searchLink> [Am J Physiol Cell Physiol] 2013 Mar; Vol. 304 (6), pp. C508-18. <i>Date of Electronic Publication: </i>2013 Jan 09. – Name: TypePub Label: Publication Type Group: TypPub Data: Journal Article; Research Support, Non-U.S. Gov't – Name: TitleSource Label: Journal Info Group: Src Data: <i>Publisher: </i><searchLink fieldCode="PB" term="%22American+Physiological+Society%22">American Physiological Society </searchLink><i>Country of Publication: </i>United States <i>NLM ID: </i>100901225 <i>Publication Model: </i>Print-Electronic <i>Cited Medium: </i>Internet <i>ISSN: </i>1522-1563 (Electronic) <i>Linking ISSN: </i><searchLink fieldCode="IS" term="%2203636143%22">03636143 </searchLink><i>NLM ISO Abbreviation: </i>Am J Physiol Cell Physiol <i>Subsets: </i>MEDLINE |
| PLink | https://search.ebscohost.com/login.aspx?direct=true&site=eds-live&db=mdl&AN=23302777 |
| RecordInfo | BibRecord: BibEntity: Identifiers: – Type: doi Value: 10.1152/ajpcell.00248.2012 Languages: – Code: eng Text: English PhysicalDescription: Pagination: StartPage: C508 Titles: – TitleFull: Exendin-4 attenuates high glucose-induced cardiomyocyte apoptosis via inhibition of endoplasmic reticulum stress and activation of SERCA2a. Type: main BibRelationships: HasContributorRelationships: – PersonEntity: Name: NameFull: Younce CW – PersonEntity: Name: NameFull: Burmeister MA – PersonEntity: Name: NameFull: Ayala JE IsPartOfRelationships: – BibEntity: Dates: – D: 01 M: 03 Text: 2013 Mar Type: published Y: 2013 Identifiers: – Type: issn-electronic Value: 1522-1563 Numbering: – Type: volume Value: 304 – Type: issue Value: 6 Titles: – TitleFull: American journal of physiology. Cell physiology Type: main |
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