[The up-regulation of hypoxia inducible factor-1α by hypoxic postconditioning reduces hypoxia/reoxygenation-induced injury in heart-derived H9c2 cells].

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Title: [The up-regulation of hypoxia inducible factor-1α by hypoxic postconditioning reduces hypoxia/reoxygenation-induced injury in heart-derived H9c2 cells].
Authors: Zhao HX; Department of Physiology, Shanxi University of Traditional Chinese Medicine, Taiyuan 030024, China; Department of Physiology, Shanxi Medical University, Taiyuan 030001, China; School of Basic Medical Sciences, and Cardiovascular Research Institute, Capital Medical University, Beijing 100069, China. E-mail: zhaohx5@126.com., Li XY, Yao H, Wu Y, Yang R, Zhao RR, Liu HR
Source: Sheng li xue bao : [Acta physiologica Sinica] [Sheng Li Xue Bao] 2013 Jun 25; Vol. 65 (3), pp. 293-300.
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
Journal Info: Publisher: Science Press Country of Publication: China NLM ID: 20730130R Publication Model: Print Cited Medium: Print ISSN: 0371-0874 (Print) Linking ISSN: 03710874 NLM ISO Abbreviation: Sheng Li Xue Bao Subsets: MEDLINE
Database: MEDLINE Ultimate
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Header DbId: mdl
DbLabel: MEDLINE Ultimate
An: 23788186
AccessLevel: 2
PubType: Academic Journal
PubTypeId: academicJournal
PreciseRelevancyScore: 0
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  Data: [The up-regulation of hypoxia inducible factor-1α by hypoxic postconditioning reduces hypoxia/reoxygenation-induced injury in heart-derived H9c2 cells].
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  Label: Authors
  Group: Au
  Data: <searchLink fieldCode="AU" term="%22Zhao+HX%22">Zhao HX</searchLink>; Department of Physiology, Shanxi University of Traditional Chinese Medicine, Taiyuan 030024, China; Department of Physiology, Shanxi Medical University, Taiyuan 030001, China; School of Basic Medical Sciences, and Cardiovascular Research Institute, Capital Medical University, Beijing 100069, China. E-mail: zhaohx5@126.com.<br /><searchLink fieldCode="AU" term="%22Li+XY%22">Li XY</searchLink><br /><searchLink fieldCode="AU" term="%22Yao+H%22">Yao H</searchLink><br /><searchLink fieldCode="AU" term="%22Wu+Y%22">Wu Y</searchLink><br /><searchLink fieldCode="AU" term="%22Yang+R%22">Yang R</searchLink><br /><searchLink fieldCode="AU" term="%22Zhao+RR%22">Zhao RR</searchLink><br /><searchLink fieldCode="AU" term="%22Liu+HR%22">Liu HR</searchLink>
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  Data: <searchLink fieldCode="JN" term="%2220730130R%22">Sheng li xue bao : [Acta physiologica Sinica]</searchLink> [Sheng Li Xue Bao] 2013 Jun 25; Vol. 65 (3), pp. 293-300.
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  Label: Publication Type
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  Data: Journal Article; Research Support, Non-U.S. Gov't
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  Data: <i>Publisher: </i><searchLink fieldCode="PB" term="%22Science+Press%22">Science Press </searchLink><i>Country of Publication: </i>China <i>NLM ID: </i>20730130R <i>Publication Model: </i>Print <i>Cited Medium: </i>Print <i>ISSN: </i>0371-0874 (Print) <i>Linking ISSN: </i><searchLink fieldCode="IS" term="%2203710874%22">03710874 </searchLink><i>NLM ISO Abbreviation: </i>Sheng Li Xue Bao <i>Subsets: </i>MEDLINE
PLink https://search.ebscohost.com/login.aspx?direct=true&site=eds-live&db=mdl&AN=23788186
RecordInfo BibRecord:
  BibEntity:
    Languages:
      – Code: chi
        Text: Chinese
    PhysicalDescription:
      Pagination:
        StartPage: 293
    Titles:
      – TitleFull: [The up-regulation of hypoxia inducible factor-1α by hypoxic postconditioning reduces hypoxia/reoxygenation-induced injury in heart-derived H9c2 cells].
        Type: main
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      – PersonEntity:
          Name:
            NameFull: Zhao HX
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            NameFull: Li XY
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            NameFull: Yao H
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            NameFull: Wu Y
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            NameFull: Yang R
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            NameFull: Zhao RR
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            NameFull: Liu HR
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          Dates:
            – D: 25
              M: 06
              Text: 2013 Jun 25
              Type: published
              Y: 2013
          Identifiers:
            – Type: issn-print
              Value: 0371-0874
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            – Type: volume
              Value: 65
            – Type: issue
              Value: 3
          Titles:
            – TitleFull: Sheng li xue bao : [Acta physiologica Sinica]
              Type: main
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