Polyglutamine-expanded androgen receptor interferes with TFEB to elicit autophagy defects in SBMA.
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| Title: | Polyglutamine-expanded androgen receptor interferes with TFEB to elicit autophagy defects in SBMA. |
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| Authors: | Cortes CJ; 1] Department of Pediatrics, University of California, San Diego, La Jolla, California, USA. [2]., Miranda HC; 1] Department of Pediatrics, University of California, San Diego, La Jolla, California, USA. [2] Department of Cellular &Molecular Medicine, University of California, San Diego, La Jolla, California, USA. [3]., Frankowski H; Department of Pediatrics, University of California, San Diego, La Jolla, California, USA., Batlevi Y; Department of Pediatrics, University of California, San Diego, La Jolla, California, USA., Young JE; Department of Cellular &Molecular Medicine, University of California, San Diego, La Jolla, California, USA., Le A; Department of Pediatrics, University of California, San Diego, La Jolla, California, USA., Ivanov N; 1] Center on Human Development &Disability, University of Washington, Seattle, Washington, USA. [2] Department of Neurology, University of Washington, Seattle, Washington, USA., Sopher BL; Department of Neurology, University of Washington, Seattle, Washington, USA., Carromeu C; Department of Pediatrics, University of California, San Diego, La Jolla, California, USA., Muotri AR; 1] Department of Pediatrics, University of California, San Diego, La Jolla, California, USA. [2] Department of Cellular &Molecular Medicine, University of California, San Diego, La Jolla, California, USA. [3] Department of Neurosciences, University of California, San Diego, La Jolla, California, USA. [4] Institute for Genomic Medicine, University of California, San Diego, La Jolla, California, USA. [5] Sanford Consortium for Regenerative Medicine, University of California, San Diego, La Jolla, California, USA. [6] Rady Children's Hospital, San Diego, California, USA., Garden GA; 1] Center on Human Development &Disability, University of Washington, Seattle, Washington, USA. [2] Department of Neurology, University of Washington, Seattle, Washington, USA., La Spada AR; 1] Department of Pediatrics, University of California, San Diego, La Jolla, California, USA. [2] Department of Cellular &Molecular Medicine, University of California, San Diego, La Jolla, California, USA. [3] Department of Neurosciences, University of California, San Diego, La Jolla, California, USA. [4] Institute for Genomic Medicine, University of California, San Diego, La Jolla, California, USA. [5] Sanford Consortium for Regenerative Medicine, University of California, San Diego, La Jolla, California, USA. [6] Rady Children's Hospital, San Diego, California, USA. [7] Division of Biological Sciences, University of California, San Diego, La Jolla, California, USA. |
| Source: | Nature neuroscience [Nat Neurosci] 2014 Sep; Vol. 17 (9), pp. 1180-9. Date of Electronic Publication: 2014 Aug 10. |
| Publication Type: | Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't |
| Journal Info: | Publisher: Nature Publishing Group Country of Publication: United States NLM ID: 9809671 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1546-1726 (Electronic) Linking ISSN: 10976256 NLM ISO Abbreviation: Nat Neurosci Subsets: MEDLINE |
| Database: | MEDLINE Ultimate |
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