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Soluble amyloid beta levels are elevated in the white matter of Alzheimer's patients, independent of cortical plaque severity.

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Title: Soluble amyloid beta levels are elevated in the white matter of Alzheimer's patients, independent of cortical plaque severity.
Authors: Collins-Praino LE, Francis YI, Griffith EY, Wiegman AF, Urbach J, Lawton A, Honig LS, Cortes E, Vonsattel JP, Canoll PD, Goldman JE, Brickman AM; Taub Institute for Research on Alzheimer's Disease and the Aging Brain, College of Physicians and Surgeons, Columbia University, 630 West 168th Street, New York 10032, NY, USA. amb2139@columbia.edu.
Source: Acta neuropathologica communications [Acta Neuropathol Commun] 2014 Aug 17; Vol. 2, pp. 83. Date of Electronic Publication: 2014 Aug 17.
Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
Journal Info: Publisher: BioMed Central Country of Publication: England NLM ID: 101610673 Publication Model: Electronic Cited Medium: Internet ISSN: 2051-5960 (Electronic) Linking ISSN: 20515960 NLM ISO Abbreviation: Acta Neuropathol Commun Subsets: MEDLINE
Database: MEDLINE Ultimate
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  Data: Soluble amyloid beta levels are elevated in the white matter of Alzheimer's patients, independent of cortical plaque severity.
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  Data: <searchLink fieldCode="AU" term="%22Collins-Praino+LE%22">Collins-Praino LE</searchLink><br /><searchLink fieldCode="AU" term="%22Francis+YI%22">Francis YI</searchLink><br /><searchLink fieldCode="AU" term="%22Griffith+EY%22">Griffith EY</searchLink><br /><searchLink fieldCode="AU" term="%22Wiegman+AF%22">Wiegman AF</searchLink><br /><searchLink fieldCode="AU" term="%22Urbach+J%22">Urbach J</searchLink><br /><searchLink fieldCode="AU" term="%22Lawton+A%22">Lawton A</searchLink><br /><searchLink fieldCode="AU" term="%22Honig+LS%22">Honig LS</searchLink><br /><searchLink fieldCode="AU" term="%22Cortes+E%22">Cortes E</searchLink><br /><searchLink fieldCode="AU" term="%22Vonsattel+JP%22">Vonsattel JP</searchLink><br /><searchLink fieldCode="AU" term="%22Canoll+PD%22">Canoll PD</searchLink><br /><searchLink fieldCode="AU" term="%22Goldman+JE%22">Goldman JE</searchLink><br /><searchLink fieldCode="AU" term="%22Brickman+AM%22">Brickman AM</searchLink>; Taub Institute for Research on Alzheimer's Disease and the Aging Brain, College of Physicians and Surgeons, Columbia University, 630 West 168th Street, New York 10032, NY, USA. amb2139@columbia.edu.
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  Data: <searchLink fieldCode="JN" term="%22101610673%22">Acta neuropathologica communications</searchLink> [Acta Neuropathol Commun] 2014 Aug 17; Vol. 2, pp. 83. <i>Date of Electronic Publication: </i>2014 Aug 17.
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  Data: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
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  Data: <i>Publisher: </i><searchLink fieldCode="PB" term="%22BioMed+Central%22">BioMed Central </searchLink><i>Country of Publication: </i>England <i>NLM ID: </i>101610673 <i>Publication Model: </i>Electronic <i>Cited Medium: </i>Internet <i>ISSN: </i>2051-5960 (Electronic) <i>Linking ISSN: </i><searchLink fieldCode="IS" term="%2220515960%22">20515960 </searchLink><i>NLM ISO Abbreviation: </i>Acta Neuropathol Commun <i>Subsets: </i>MEDLINE
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        Value: 10.1186/s40478-014-0083-0
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              Text: 2014 Aug 17
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