Tetrandrine induces programmed cell death in human oral cancer CAL 27 cells through the reactive oxygen species production and caspase-dependent pathways and associated with beclin-1-induced cell autophagy.

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Title: Tetrandrine induces programmed cell death in human oral cancer CAL 27 cells through the reactive oxygen species production and caspase-dependent pathways and associated with beclin-1-induced cell autophagy.
Authors: Lien JC; Graduate Institute of Pharmaceutical Chemistry, China Medical University, Taichung, 404, Taiwan., Lin MW; Department of Nursing, Cardinal Tien Junior College of Healthcare and Management, New Taipei, 231, Taiwan., Chang SJ; School of Pharmacy, China Medical University, Taichung, 404, Taiwan., Lai KC; Department of Surgery, China Medical University Beigang Hospital, Yunlin, 651, Taiwan.; School of Medicine, China Medical University Hospital, Taichung, 404, Taiwan., Huang AC; Department of Nursing, St. Mary's Medicine Nursing and Management College, Yilan, 266, Taiwan., Yu FS; Department of Dentist, China Medical University, Taichung, 404, Taiwan., Chung JG; Department of Biological Science and Technology, China Medical University, Taichung, 404, Taiwan.; Department of Biotechnology, Asia University, Taichung, 413, Taiwan.
Source: Environmental toxicology [Environ Toxicol] 2017 Jan; Vol. 32 (1), pp. 329-343. Date of Electronic Publication: 2016 Jan 29.
Publication Type: Journal Article
Journal Info: Publisher: John Wiley & Sons Country of Publication: United States NLM ID: 100885357 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1522-7278 (Electronic) Linking ISSN: 15204081 NLM ISO Abbreviation: Environ Toxicol Subsets: MEDLINE
Database: MEDLINE Ultimate
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  Data: Tetrandrine induces programmed cell death in human oral cancer CAL 27 cells through the reactive oxygen species production and caspase-dependent pathways and associated with beclin-1-induced cell autophagy.
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  Data: <searchLink fieldCode="AU" term="%22Lien+JC%22">Lien JC</searchLink>; Graduate Institute of Pharmaceutical Chemistry, China Medical University, Taichung, 404, Taiwan.<br /><searchLink fieldCode="AU" term="%22Lin+MW%22">Lin MW</searchLink>; Department of Nursing, Cardinal Tien Junior College of Healthcare and Management, New Taipei, 231, Taiwan.<br /><searchLink fieldCode="AU" term="%22Chang+SJ%22">Chang SJ</searchLink>; School of Pharmacy, China Medical University, Taichung, 404, Taiwan.<br /><searchLink fieldCode="AU" term="%22Lai+KC%22">Lai KC</searchLink>; Department of Surgery, China Medical University Beigang Hospital, Yunlin, 651, Taiwan.; School of Medicine, China Medical University Hospital, Taichung, 404, Taiwan.<br /><searchLink fieldCode="AU" term="%22Huang+AC%22">Huang AC</searchLink>; Department of Nursing, St. Mary's Medicine Nursing and Management College, Yilan, 266, Taiwan.<br /><searchLink fieldCode="AU" term="%22Yu+FS%22">Yu FS</searchLink>; Department of Dentist, China Medical University, Taichung, 404, Taiwan.<br /><searchLink fieldCode="AU" term="%22Chung+JG%22">Chung JG</searchLink>; Department of Biological Science and Technology, China Medical University, Taichung, 404, Taiwan.; Department of Biotechnology, Asia University, Taichung, 413, Taiwan.
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  Data: <searchLink fieldCode="JN" term="%22100885357%22">Environmental toxicology</searchLink> [Environ Toxicol] 2017 Jan; Vol. 32 (1), pp. 329-343. <i>Date of Electronic Publication: </i>2016 Jan 29.
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  Data: <i>Publisher: </i><searchLink fieldCode="PB" term="%22John+Wiley+%26+Sons%22">John Wiley & Sons </searchLink><i>Country of Publication: </i>United States <i>NLM ID: </i>100885357 <i>Publication Model: </i>Print-Electronic <i>Cited Medium: </i>Internet <i>ISSN: </i>1522-7278 (Electronic) <i>Linking ISSN: </i><searchLink fieldCode="IS" term="%2215204081%22">15204081 </searchLink><i>NLM ISO Abbreviation: </i>Environ Toxicol <i>Subsets: </i>MEDLINE
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        Value: 10.1002/tox.22238
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      – TitleFull: Tetrandrine induces programmed cell death in human oral cancer CAL 27 cells through the reactive oxygen species production and caspase-dependent pathways and associated with beclin-1-induced cell autophagy.
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