Intracrine angiotensin II functions originate from noncanonical pathways in the human heart.

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Title: Intracrine angiotensin II functions originate from noncanonical pathways in the human heart.
Authors: Ferrario CM; Departments of Surgery, Internal Medicine-Nephrology and Physiology-Pharmacology, Wake Forest University Health Science Center, Winston-Salem, North Carolina; cferrari@wakehealth.edu., Ahmad S; Departments of Surgery, Internal Medicine-Nephrology and Physiology-Pharmacology, Wake Forest University Health Science Center, Winston-Salem, North Carolina;, Varagic J; Departments of Surgery, Internal Medicine-Nephrology and Physiology-Pharmacology, Wake Forest University Health Science Center, Winston-Salem, North Carolina; Hypertension and Vascular Research Center, Wake Forest University Health Science Center, Winston-Salem, North Carolina;, Cheng CP; Section on Cardiovascular Medicine, Department of Internal Medicine, Wake Forest University Health Science Center, Winston-Salem, North Carolina;, Groban L; Hypertension and Vascular Research Center, Wake Forest University Health Science Center, Winston-Salem, North Carolina; Department of Anesthesiology, Wake Forest University Health Science Center, Winston-Salem, North Carolina;, Wang H; Department of Anesthesiology, Wake Forest University Health Science Center, Winston-Salem, North Carolina;, Collawn JF; Departments of Cell Biology, Microbiology, Physiology, University of Alabama Birmingham, Alabama; and., Dell Italia LJ; Departments of Cell Biology, Microbiology, Physiology, University of Alabama Birmingham, Alabama; and Division of Cardiovascular Disease, University of Alabama at Birmingham and Department of Veterans Affairs, Birmingham Veterans Affairs Medical Center, Birmingham, Alabama.
Source: American journal of physiology. Heart and circulatory physiology [Am J Physiol Heart Circ Physiol] 2016 Aug 01; Vol. 311 (2), pp. H404-14. Date of Electronic Publication: 2016 May 27.
Publication Type: Journal Article; Review; Research Support, N.I.H., Extramural
Journal Info: Publisher: American Physiological Society Country of Publication: United States NLM ID: 100901228 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1522-1539 (Electronic) Linking ISSN: 03636135 NLM ISO Abbreviation: Am J Physiol Heart Circ Physiol Subsets: MEDLINE
Database: MEDLINE Ultimate
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  Data: Intracrine angiotensin II functions originate from noncanonical pathways in the human heart.
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  Data: <searchLink fieldCode="AU" term="%22Ferrario+CM%22">Ferrario CM</searchLink>; Departments of Surgery, Internal Medicine-Nephrology and Physiology-Pharmacology, Wake Forest University Health Science Center, Winston-Salem, North Carolina; cferrari@wakehealth.edu.<br /><searchLink fieldCode="AU" term="%22Ahmad+S%22">Ahmad S</searchLink>; Departments of Surgery, Internal Medicine-Nephrology and Physiology-Pharmacology, Wake Forest University Health Science Center, Winston-Salem, North Carolina;<br /><searchLink fieldCode="AU" term="%22Varagic+J%22">Varagic J</searchLink>; Departments of Surgery, Internal Medicine-Nephrology and Physiology-Pharmacology, Wake Forest University Health Science Center, Winston-Salem, North Carolina; Hypertension and Vascular Research Center, Wake Forest University Health Science Center, Winston-Salem, North Carolina;<br /><searchLink fieldCode="AU" term="%22Cheng+CP%22">Cheng CP</searchLink>; Section on Cardiovascular Medicine, Department of Internal Medicine, Wake Forest University Health Science Center, Winston-Salem, North Carolina;<br /><searchLink fieldCode="AU" term="%22Groban+L%22">Groban L</searchLink>; Hypertension and Vascular Research Center, Wake Forest University Health Science Center, Winston-Salem, North Carolina; Department of Anesthesiology, Wake Forest University Health Science Center, Winston-Salem, North Carolina;<br /><searchLink fieldCode="AU" term="%22Wang+H%22">Wang H</searchLink>; Department of Anesthesiology, Wake Forest University Health Science Center, Winston-Salem, North Carolina;<br /><searchLink fieldCode="AU" term="%22Collawn+JF%22">Collawn JF</searchLink>; Departments of Cell Biology, Microbiology, Physiology, University of Alabama Birmingham, Alabama; and.<br /><searchLink fieldCode="AU" term="%22Dell+Italia+LJ%22">Dell Italia LJ</searchLink>; Departments of Cell Biology, Microbiology, Physiology, University of Alabama Birmingham, Alabama; and Division of Cardiovascular Disease, University of Alabama at Birmingham and Department of Veterans Affairs, Birmingham Veterans Affairs Medical Center, Birmingham, Alabama.
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  Data: <searchLink fieldCode="JN" term="%22100901228%22">American journal of physiology. Heart and circulatory physiology</searchLink> [Am J Physiol Heart Circ Physiol] 2016 Aug 01; Vol. 311 (2), pp. H404-14. <i>Date of Electronic Publication: </i>2016 May 27.
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  Data: <i>Publisher: </i><searchLink fieldCode="PB" term="%22American+Physiological+Society%22">American Physiological Society </searchLink><i>Country of Publication: </i>United States <i>NLM ID: </i>100901228 <i>Publication Model: </i>Print-Electronic <i>Cited Medium: </i>Internet <i>ISSN: </i>1522-1539 (Electronic) <i>Linking ISSN: </i><searchLink fieldCode="IS" term="%2203636135%22">03636135 </searchLink><i>NLM ISO Abbreviation: </i>Am J Physiol Heart Circ Physiol <i>Subsets: </i>MEDLINE
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        Value: 10.1152/ajpheart.00219.2016
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        Text: English
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              Text: 2016 Aug 01
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