APA (7th ed.) Citation

CH, S., CL, K., KW, L., FS, Y., YS, M., JL, Y., . . . JG, C. (2017). Fisetin-induced apoptosis of human oral cancer SCC-4 cells through reactive oxygen species production, endoplasmic reticulum stress, caspase-, and mitochondria-dependent signaling pathways. Environmental toxicology, 32(6), 1725. https://doi.org/10.1002/tox.22396

Chicago Style (17th ed.) Citation

CH, Su, et al. "Fisetin-induced Apoptosis of Human Oral Cancer SCC-4 Cells Through Reactive Oxygen Species Production, Endoplasmic Reticulum Stress, Caspase-, and Mitochondria-dependent Signaling Pathways." Environmental Toxicology 32, no. 6 (2017): 1725. https://doi.org/10.1002/tox.22396.

MLA (9th ed.) Citation

CH, Su, et al. "Fisetin-induced Apoptosis of Human Oral Cancer SCC-4 Cells Through Reactive Oxygen Species Production, Endoplasmic Reticulum Stress, Caspase-, and Mitochondria-dependent Signaling Pathways." Environmental Toxicology, vol. 32, no. 6, 2017, p. 1725, https://doi.org/10.1002/tox.22396.