Adhesion-induced eosinophil cytolysis requires the receptor-interacting protein kinase 3 (RIPK3)-mixed lineage kinase-like (MLKL) signaling pathway, which is counterregulated by autophagy.

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Title: Adhesion-induced eosinophil cytolysis requires the receptor-interacting protein kinase 3 (RIPK3)-mixed lineage kinase-like (MLKL) signaling pathway, which is counterregulated by autophagy.
Authors: Radonjic-Hoesli S; Institute of Pharmacology, University of Bern, Bern, Switzerland; Department of Dermatology, Inselspital, Bern University Hospital, University of Bern, Bern, Switzerland., Wang X; Institute of Pharmacology, University of Bern, Bern, Switzerland., de Graauw E; Institute of Pharmacology, University of Bern, Bern, Switzerland., Stoeckle C; Institute of Pharmacology, University of Bern, Bern, Switzerland., Styp-Rekowska B; Institute of Anatomy, University of Bern, Bern, Switzerland., Hlushchuk R; Institute of Anatomy, University of Bern, Bern, Switzerland., Simon D; Department of Dermatology, Inselspital, Bern University Hospital, University of Bern, Bern, Switzerland., Spaeth PJ; Institute of Pharmacology, University of Bern, Bern, Switzerland., Yousefi S; Institute of Pharmacology, University of Bern, Bern, Switzerland., Simon HU; Institute of Pharmacology, University of Bern, Bern, Switzerland. Electronic address: hus@pki.unibe.ch.
Source: The Journal of allergy and clinical immunology [J Allergy Clin Immunol] 2017 Dec; Vol. 140 (6), pp. 1632-1642. Date of Electronic Publication: 2017 Apr 12.
Publication Type: Journal Article
Journal Info: Publisher: Mosby Country of Publication: United States NLM ID: 1275002 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1097-6825 (Electronic) Linking ISSN: 00916749 NLM ISO Abbreviation: J Allergy Clin Immunol Subsets: MEDLINE
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  Data: Adhesion-induced eosinophil cytolysis requires the receptor-interacting protein kinase 3 (RIPK3)-mixed lineage kinase-like (MLKL) signaling pathway, which is counterregulated by autophagy.
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  Data: <searchLink fieldCode="AU" term="%22Radonjic-Hoesli+S%22">Radonjic-Hoesli S</searchLink>; Institute of Pharmacology, University of Bern, Bern, Switzerland; Department of Dermatology, Inselspital, Bern University Hospital, University of Bern, Bern, Switzerland.<br /><searchLink fieldCode="AU" term="%22Wang+X%22">Wang X</searchLink>; Institute of Pharmacology, University of Bern, Bern, Switzerland.<br /><searchLink fieldCode="AU" term="%22de+Graauw+E%22">de Graauw E</searchLink>; Institute of Pharmacology, University of Bern, Bern, Switzerland.<br /><searchLink fieldCode="AU" term="%22Stoeckle+C%22">Stoeckle C</searchLink>; Institute of Pharmacology, University of Bern, Bern, Switzerland.<br /><searchLink fieldCode="AU" term="%22Styp-Rekowska+B%22">Styp-Rekowska B</searchLink>; Institute of Anatomy, University of Bern, Bern, Switzerland.<br /><searchLink fieldCode="AU" term="%22Hlushchuk+R%22">Hlushchuk R</searchLink>; Institute of Anatomy, University of Bern, Bern, Switzerland.<br /><searchLink fieldCode="AU" term="%22Simon+D%22">Simon D</searchLink>; Department of Dermatology, Inselspital, Bern University Hospital, University of Bern, Bern, Switzerland.<br /><searchLink fieldCode="AU" term="%22Spaeth+PJ%22">Spaeth PJ</searchLink>; Institute of Pharmacology, University of Bern, Bern, Switzerland.<br /><searchLink fieldCode="AU" term="%22Yousefi+S%22">Yousefi S</searchLink>; Institute of Pharmacology, University of Bern, Bern, Switzerland.<br /><searchLink fieldCode="AU" term="%22Simon+HU%22">Simon HU</searchLink>; Institute of Pharmacology, University of Bern, Bern, Switzerland. Electronic address: hus@pki.unibe.ch.
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  Data: <i>Publisher: </i><searchLink fieldCode="PB" term="%22Mosby%22">Mosby </searchLink><i>Country of Publication: </i>United States <i>NLM ID: </i>1275002 <i>Publication Model: </i>Print-Electronic <i>Cited Medium: </i>Internet <i>ISSN: </i>1097-6825 (Electronic) <i>Linking ISSN: </i><searchLink fieldCode="IS" term="%2200916749%22">00916749 </searchLink><i>NLM ISO Abbreviation: </i>J Allergy Clin Immunol <i>Subsets: </i>MEDLINE
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      – TitleFull: Adhesion-induced eosinophil cytolysis requires the receptor-interacting protein kinase 3 (RIPK3)-mixed lineage kinase-like (MLKL) signaling pathway, which is counterregulated by autophagy.
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              Text: 2017 Dec
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