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Replication fork slowing and stalling are distinct, checkpoint-independent consequences of replicating damaged DNA.

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Title: Replication fork slowing and stalling are distinct, checkpoint-independent consequences of replicating damaged DNA.
Authors: Iyer DR; Department of Biochemistry & Molecular Pharmacology, University of Massachusetts Medical School, Worcester, Massachusetts, United States of America., Rhind N; Department of Biochemistry & Molecular Pharmacology, University of Massachusetts Medical School, Worcester, Massachusetts, United States of America.
Source: PLoS genetics [PLoS Genet] 2017 Aug 14; Vol. 13 (8), pp. e1006958. Date of Electronic Publication: 2017 Aug 14 (Print Publication: 2017).
Publication Type: Journal Article
Journal Info: Publisher: Public Library of Science Country of Publication: United States NLM ID: 101239074 Publication Model: eCollection Cited Medium: Internet ISSN: 1553-7404 (Electronic) Linking ISSN: 15537390 NLM ISO Abbreviation: PLoS Genet Subsets: MEDLINE
Database: MEDLINE Ultimate
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  Data: <searchLink fieldCode="AU" term="%22Iyer+DR%22">Iyer DR</searchLink>; Department of Biochemistry & Molecular Pharmacology, University of Massachusetts Medical School, Worcester, Massachusetts, United States of America.<br /><searchLink fieldCode="AU" term="%22Rhind+N%22">Rhind N</searchLink>; Department of Biochemistry & Molecular Pharmacology, University of Massachusetts Medical School, Worcester, Massachusetts, United States of America.
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        Value: 10.1371/journal.pgen.1006958
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      – TitleFull: Replication fork slowing and stalling are distinct, checkpoint-independent consequences of replicating damaged DNA.
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