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Inotropic myocardial reserve deficiency is the predominant feature of exercise haemodynamics in cardiac amyloidosis.

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Title: Inotropic myocardial reserve deficiency is the predominant feature of exercise haemodynamics in cardiac amyloidosis.
Authors: Clemmensen TS; Department of Cardiology, Aarhus University Hospital, Aarhus, Denmark., Mølgaard H; Department of Cardiology, Aarhus University Hospital, Aarhus, Denmark., Sörensen J; Department of Nuclear Medicine and PET Centre, Aarhus University Hospital, Aarhus, Denmark., Eiskjaer H; Department of Cardiology, Aarhus University Hospital, Aarhus, Denmark., Andersen NF; Department of Haematology, Aarhus University Hospital, Aarhus, Denmark., Mellemkjaer S; Department of Cardiology, Aarhus University Hospital, Aarhus, Denmark., Andersen MJ; Department of Cardiology, Aarhus University Hospital, Aarhus, Denmark., Tolbod LP; Department of Nuclear Medicine and PET Centre, Aarhus University Hospital, Aarhus, Denmark., Harms HJ; Department of Nuclear Medicine and PET Centre, Aarhus University Hospital, Aarhus, Denmark., Poulsen SH; Department of Cardiology, Aarhus University Hospital, Aarhus, Denmark.
Source: European journal of heart failure [Eur J Heart Fail] 2017 Nov; Vol. 19 (11), pp. 1457-1465. Date of Electronic Publication: 2017 Aug 24.
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
Journal Info: Publisher: Wiley Country of Publication: England NLM ID: 100887595 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1879-0844 (Electronic) Linking ISSN: 13889842 NLM ISO Abbreviation: Eur J Heart Fail Subsets: MEDLINE
Database: MEDLINE Ultimate
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  Data: Inotropic myocardial reserve deficiency is the predominant feature of exercise haemodynamics in cardiac amyloidosis.
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  Data: <searchLink fieldCode="AU" term="%22Clemmensen+TS%22">Clemmensen TS</searchLink>; Department of Cardiology, Aarhus University Hospital, Aarhus, Denmark.<br /><searchLink fieldCode="AU" term="%22Mølgaard+H%22">Mølgaard H</searchLink>; Department of Cardiology, Aarhus University Hospital, Aarhus, Denmark.<br /><searchLink fieldCode="AU" term="%22Sörensen+J%22">Sörensen J</searchLink>; Department of Nuclear Medicine and PET Centre, Aarhus University Hospital, Aarhus, Denmark.<br /><searchLink fieldCode="AU" term="%22Eiskjaer+H%22">Eiskjaer H</searchLink>; Department of Cardiology, Aarhus University Hospital, Aarhus, Denmark.<br /><searchLink fieldCode="AU" term="%22Andersen+NF%22">Andersen NF</searchLink>; Department of Haematology, Aarhus University Hospital, Aarhus, Denmark.<br /><searchLink fieldCode="AU" term="%22Mellemkjaer+S%22">Mellemkjaer S</searchLink>; Department of Cardiology, Aarhus University Hospital, Aarhus, Denmark.<br /><searchLink fieldCode="AU" term="%22Andersen+MJ%22">Andersen MJ</searchLink>; Department of Cardiology, Aarhus University Hospital, Aarhus, Denmark.<br /><searchLink fieldCode="AU" term="%22Tolbod+LP%22">Tolbod LP</searchLink>; Department of Nuclear Medicine and PET Centre, Aarhus University Hospital, Aarhus, Denmark.<br /><searchLink fieldCode="AU" term="%22Harms+HJ%22">Harms HJ</searchLink>; Department of Nuclear Medicine and PET Centre, Aarhus University Hospital, Aarhus, Denmark.<br /><searchLink fieldCode="AU" term="%22Poulsen+SH%22">Poulsen SH</searchLink>; Department of Cardiology, Aarhus University Hospital, Aarhus, Denmark.
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  Data: <searchLink fieldCode="JN" term="%22100887595%22">European journal of heart failure</searchLink> [Eur J Heart Fail] 2017 Nov; Vol. 19 (11), pp. 1457-1465. <i>Date of Electronic Publication: </i>2017 Aug 24.
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  Data: <i>Publisher: </i><searchLink fieldCode="PB" term="%22Wiley%22">Wiley </searchLink><i>Country of Publication: </i>England <i>NLM ID: </i>100887595 <i>Publication Model: </i>Print-Electronic <i>Cited Medium: </i>Internet <i>ISSN: </i>1879-0844 (Electronic) <i>Linking ISSN: </i><searchLink fieldCode="IS" term="%2213889842%22">13889842 </searchLink><i>NLM ISO Abbreviation: </i>Eur J Heart Fail <i>Subsets: </i>MEDLINE
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        Value: 10.1002/ejhf.899
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              Text: 2017 Nov
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