APA (7th ed.) Citation

MBN, G., AH, S., MF, P., LS, G., A, I., H, B., . . . MM, R. (2018). Human GIP(3-30)NH2 inhibits G protein-dependent as well as G protein-independent signaling and is selective for the GIP receptor with high-affinity binding to primate but not rodent GIP receptors. Biochemical pharmacology, 150, 97. https://doi.org/10.1016/j.bcp.2018.01.040

Chicago Style (17th ed.) Citation

MBN, Gabe, Sparre-Ulrich AH, Pedersen MF, Gasbjerg LS, Inoue A, Bräuner-Osborne H, Hartmann B, and Rosenkilde MM. "Human GIP(3-30)NH2 Inhibits G Protein-dependent as Well as G Protein-independent Signaling and Is Selective for the GIP Receptor with High-affinity Binding to Primate but Not Rodent GIP Receptors." Biochemical Pharmacology 150 (2018): 97. https://doi.org/10.1016/j.bcp.2018.01.040.

MLA (9th ed.) Citation

MBN, Gabe, et al. "Human GIP(3-30)NH2 Inhibits G Protein-dependent as Well as G Protein-independent Signaling and Is Selective for the GIP Receptor with High-affinity Binding to Primate but Not Rodent GIP Receptors." Biochemical Pharmacology, vol. 150, 2018, p. 97, https://doi.org/10.1016/j.bcp.2018.01.040.