APA (7th ed.) Citation

RC, R., B, S., M, M., C, B., G, G., A, A., . . . MM, T. (2018). The atypical chemokine receptor ACKR2 drives pulmonary fibrosis by tuning influx of CCR2+ and CCR5+ IFNγ-producing γδT cells in mice. American journal of physiology. Lung cellular and molecular physiology, 314(6), L1010. https://doi.org/10.1152/ajplung.00233.2017

Chicago Style (17th ed.) Citation

RC, Russo, et al. "The Atypical Chemokine Receptor ACKR2 Drives Pulmonary Fibrosis by Tuning Influx of CCR2+ and CCR5+ IFNγ-producing γδT Cells in Mice." American Journal of Physiology. Lung Cellular and Molecular Physiology 314, no. 6 (2018): L1010. https://doi.org/10.1152/ajplung.00233.2017.

MLA (9th ed.) Citation

RC, Russo, et al. "The Atypical Chemokine Receptor ACKR2 Drives Pulmonary Fibrosis by Tuning Influx of CCR2+ and CCR5+ IFNγ-producing γδT Cells in Mice." American Journal of Physiology. Lung Cellular and Molecular Physiology, vol. 314, no. 6, 2018, p. L1010, https://doi.org/10.1152/ajplung.00233.2017.