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The atypical chemokine receptor ACKR2 drives pulmonary fibrosis by tuning influx of CCR2+ and CCR5+ IFNγ-producing γδT cells in mice.

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Title: The atypical chemokine receptor ACKR2 drives pulmonary fibrosis by tuning influx of CCR2+ and CCR5+ IFNγ-producing γδT cells in mice.
Authors: Russo RC; Laboratory of Pulmonary Immunology and Mechanics, Department of Physiology and Biophysics, Institute of Biological Sciences, Universidade Federal de Minas Gerais , Belo Horizonte , Brazil.; Laboratory of Immunopharmacology, Department of Biochemistry and Immunology, Institute of Biological Sciences, Universidade Federal de Minas Gerais , Belo Horizonte , Brazil.; Humanitas Clinical and Research Center, Rozzano, Italy., Savino B; Humanitas Clinical and Research Center, Rozzano, Italy.; Department of Medical Biotechnology and Translational Medicine, University of Milan , Milan , Italy., Mirolo M; Humanitas Clinical and Research Center, Rozzano, Italy., Buracchi C; Humanitas Clinical and Research Center, Rozzano, Italy., Germano G; Humanitas Clinical and Research Center, Rozzano, Italy., Anselmo A; Humanitas Clinical and Research Center, Rozzano, Italy., Zammataro L; Humanitas Clinical and Research Center, Rozzano, Italy., Pasqualini F; Humanitas Clinical and Research Center, Rozzano, Italy., Mantovani A; Humanitas Clinical and Research Center, Rozzano, Italy.; Humanitas University, Rozzano, Italy., Locati M; Humanitas Clinical and Research Center, Rozzano, Italy.; Department of Medical Biotechnology and Translational Medicine, University of Milan , Milan , Italy., Teixeira MM; Laboratory of Immunopharmacology, Department of Biochemistry and Immunology, Institute of Biological Sciences, Universidade Federal de Minas Gerais , Belo Horizonte , Brazil.
Source: American journal of physiology. Lung cellular and molecular physiology [Am J Physiol Lung Cell Mol Physiol] 2018 Jun 01; Vol. 314 (6), pp. L1010-L1025. Date of Electronic Publication: 2018 Feb 22.
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
Journal Info: Publisher: American Physiological Society Country of Publication: United States NLM ID: 100901229 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1522-1504 (Electronic) Linking ISSN: 10400605 NLM ISO Abbreviation: Am J Physiol Lung Cell Mol Physiol Subsets: MEDLINE
Database: MEDLINE Ultimate
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  Data: The atypical chemokine receptor ACKR2 drives pulmonary fibrosis by tuning influx of CCR2<superscript>+</superscript> and CCR5<superscript>+</superscript> IFNγ-producing γδT cells in mice.
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  Data: <searchLink fieldCode="AU" term="%22Russo+RC%22">Russo RC</searchLink>; Laboratory of Pulmonary Immunology and Mechanics, Department of Physiology and Biophysics, Institute of Biological Sciences, Universidade Federal de Minas Gerais , Belo Horizonte , Brazil.; Laboratory of Immunopharmacology, Department of Biochemistry and Immunology, Institute of Biological Sciences, Universidade Federal de Minas Gerais , Belo Horizonte , Brazil.; Humanitas Clinical and Research Center, Rozzano, Italy.<br /><searchLink fieldCode="AU" term="%22Savino+B%22">Savino B</searchLink>; Humanitas Clinical and Research Center, Rozzano, Italy.; Department of Medical Biotechnology and Translational Medicine, University of Milan , Milan , Italy.<br /><searchLink fieldCode="AU" term="%22Mirolo+M%22">Mirolo M</searchLink>; Humanitas Clinical and Research Center, Rozzano, Italy.<br /><searchLink fieldCode="AU" term="%22Buracchi+C%22">Buracchi C</searchLink>; Humanitas Clinical and Research Center, Rozzano, Italy.<br /><searchLink fieldCode="AU" term="%22Germano+G%22">Germano G</searchLink>; Humanitas Clinical and Research Center, Rozzano, Italy.<br /><searchLink fieldCode="AU" term="%22Anselmo+A%22">Anselmo A</searchLink>; Humanitas Clinical and Research Center, Rozzano, Italy.<br /><searchLink fieldCode="AU" term="%22Zammataro+L%22">Zammataro L</searchLink>; Humanitas Clinical and Research Center, Rozzano, Italy.<br /><searchLink fieldCode="AU" term="%22Pasqualini+F%22">Pasqualini F</searchLink>; Humanitas Clinical and Research Center, Rozzano, Italy.<br /><searchLink fieldCode="AU" term="%22Mantovani+A%22">Mantovani A</searchLink>; Humanitas Clinical and Research Center, Rozzano, Italy.; Humanitas University, Rozzano, Italy.<br /><searchLink fieldCode="AU" term="%22Locati+M%22">Locati M</searchLink>; Humanitas Clinical and Research Center, Rozzano, Italy.; Department of Medical Biotechnology and Translational Medicine, University of Milan , Milan , Italy.<br /><searchLink fieldCode="AU" term="%22Teixeira+MM%22">Teixeira MM</searchLink>; Laboratory of Immunopharmacology, Department of Biochemistry and Immunology, Institute of Biological Sciences, Universidade Federal de Minas Gerais , Belo Horizonte , Brazil.
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  Data: <searchLink fieldCode="JN" term="%22100901229%22">American journal of physiology. Lung cellular and molecular physiology</searchLink> [Am J Physiol Lung Cell Mol Physiol] 2018 Jun 01; Vol. 314 (6), pp. L1010-L1025. <i>Date of Electronic Publication: </i>2018 Feb 22.
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  Data: <i>Publisher: </i><searchLink fieldCode="PB" term="%22American+Physiological+Society%22">American Physiological Society </searchLink><i>Country of Publication: </i>United States <i>NLM ID: </i>100901229 <i>Publication Model: </i>Print-Electronic <i>Cited Medium: </i>Internet <i>ISSN: </i>1522-1504 (Electronic) <i>Linking ISSN: </i><searchLink fieldCode="IS" term="%2210400605%22">10400605 </searchLink><i>NLM ISO Abbreviation: </i>Am J Physiol Lung Cell Mol Physiol <i>Subsets: </i>MEDLINE
PLink https://search.ebscohost.com/login.aspx?direct=true&site=eds-live&db=mdl&AN=29469612
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              Text: 2018 Jun 01
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