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Functional disruption of the Golgi apparatus protein ARF1 sensitizes MDA-MB-231 breast cancer cells to the antitumor drugs Actinomycin D and Vinblastine through ERK and AKT signaling.

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Title: Functional disruption of the Golgi apparatus protein ARF1 sensitizes MDA-MB-231 breast cancer cells to the antitumor drugs Actinomycin D and Vinblastine through ERK and AKT signaling.
Authors: Luchsinger C; Department of Physiology, School of Medicine, Universidad Austral de Chile, Valdivia, Chile.; Center for Interdisciplinary Studies of the Nervous System (CISNe), Universidad Austral de Chile, Valdivia, Chile., Aguilar M; Department of Physiology, School of Medicine, Universidad Austral de Chile, Valdivia, Chile.; Center for Interdisciplinary Studies of the Nervous System (CISNe), Universidad Austral de Chile, Valdivia, Chile., Burgos PV; Department of Physiology, School of Medicine, Universidad Austral de Chile, Valdivia, Chile.; Center for Interdisciplinary Studies of the Nervous System (CISNe), Universidad Austral de Chile, Valdivia, Chile.; Center for Cell Biology and Biomedicine (CEBICEM), School of Medicine and Science, Universidad San Sebastián, Santiago, Chile.; Center for Aging and Regeneration (CARE), Facultad de Ciencias Biológicas, Pontificia Universidad Católica de Chile, Santiago, Chile., Ehrenfeld P; Center for Interdisciplinary Studies of the Nervous System (CISNe), Universidad Austral de Chile, Valdivia, Chile.; Department of Anatomy, Histology and Pathology, School of Medicine, Universidad Austral de Chile, Valdivia, Chile., Mardones GA; Department of Physiology, School of Medicine, Universidad Austral de Chile, Valdivia, Chile.; Center for Interdisciplinary Studies of the Nervous System (CISNe), Universidad Austral de Chile, Valdivia, Chile.; Center for Cell Biology and Biomedicine (CEBICEM), School of Medicine and Science, Universidad San Sebastián, Santiago, Chile.
Source: PloS one [PLoS One] 2018 Apr 03; Vol. 13 (4), pp. e0195401. Date of Electronic Publication: 2018 Apr 03 (Print Publication: 2018).
Publication Type: Journal Article
Journal Info: Publisher: Public Library of Science Country of Publication: United States NLM ID: 101285081 Publication Model: eCollection Cited Medium: Internet ISSN: 1932-6203 (Electronic) Linking ISSN: 19326203 NLM ISO Abbreviation: PLoS One Subsets: MEDLINE
Database: MEDLINE Ultimate
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  Data: Functional disruption of the Golgi apparatus protein ARF1 sensitizes MDA-MB-231 breast cancer cells to the antitumor drugs Actinomycin D and Vinblastine through ERK and AKT signaling.
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  Data: <searchLink fieldCode="AU" term="%22Luchsinger+C%22">Luchsinger C</searchLink>; Department of Physiology, School of Medicine, Universidad Austral de Chile, Valdivia, Chile.; Center for Interdisciplinary Studies of the Nervous System (CISNe), Universidad Austral de Chile, Valdivia, Chile.<br /><searchLink fieldCode="AU" term="%22Aguilar+M%22">Aguilar M</searchLink>; Department of Physiology, School of Medicine, Universidad Austral de Chile, Valdivia, Chile.; Center for Interdisciplinary Studies of the Nervous System (CISNe), Universidad Austral de Chile, Valdivia, Chile.<br /><searchLink fieldCode="AU" term="%22Burgos+PV%22">Burgos PV</searchLink>; Department of Physiology, School of Medicine, Universidad Austral de Chile, Valdivia, Chile.; Center for Interdisciplinary Studies of the Nervous System (CISNe), Universidad Austral de Chile, Valdivia, Chile.; Center for Cell Biology and Biomedicine (CEBICEM), School of Medicine and Science, Universidad San Sebastián, Santiago, Chile.; Center for Aging and Regeneration (CARE), Facultad de Ciencias Biológicas, Pontificia Universidad Católica de Chile, Santiago, Chile.<br /><searchLink fieldCode="AU" term="%22Ehrenfeld+P%22">Ehrenfeld P</searchLink>; Center for Interdisciplinary Studies of the Nervous System (CISNe), Universidad Austral de Chile, Valdivia, Chile.; Department of Anatomy, Histology and Pathology, School of Medicine, Universidad Austral de Chile, Valdivia, Chile.<br /><searchLink fieldCode="AU" term="%22Mardones+GA%22">Mardones GA</searchLink>; Department of Physiology, School of Medicine, Universidad Austral de Chile, Valdivia, Chile.; Center for Interdisciplinary Studies of the Nervous System (CISNe), Universidad Austral de Chile, Valdivia, Chile.; Center for Cell Biology and Biomedicine (CEBICEM), School of Medicine and Science, Universidad San Sebastián, Santiago, Chile.
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