Inhibition of FOXO1 transcription factor in primary human adipocytes mimics the insulin-resistant state of type 2 diabetes.

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Title: Inhibition of FOXO1 transcription factor in primary human adipocytes mimics the insulin-resistant state of type 2 diabetes.
Authors: Rajan MR; Department of Clinical and Experimental Medicine, Linköping University, SE-58185 Linköping, Sweden., Nyman E; Department of Biomedical Engineering, Linköping University, SE-58185 Linköping, Sweden., Brännmark C; Department of Neuroscience and Physiology, Metabolic Physiology Section, University of Gothenburg, SE-41390 Göteborg, Sweden., Olofsson CS; Department of Neuroscience and Physiology, Metabolic Physiology Section, University of Gothenburg, SE-41390 Göteborg, Sweden., Strålfors P; Department of Clinical and Experimental Medicine, Linköping University, SE-58185 Linköping, Sweden peter.stralfors@liu.se.
Source: The Biochemical journal [Biochem J] 2018 May 31; Vol. 475 (10), pp. 1807-1820. Date of Electronic Publication: 2018 May 31.
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
Journal Info: Publisher: Published by Portland Press on behalf of the Biochemical Society Country of Publication: England NLM ID: 2984726R Publication Model: Electronic Cited Medium: Internet ISSN: 1470-8728 (Electronic) Linking ISSN: 02646021 NLM ISO Abbreviation: Biochem J Subsets: MEDLINE
Database: MEDLINE Ultimate
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  Data: Inhibition of FOXO1 transcription factor in primary human adipocytes mimics the insulin-resistant state of type 2 diabetes.
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  Data: <searchLink fieldCode="AU" term="%22Rajan+MR%22">Rajan MR</searchLink>; Department of Clinical and Experimental Medicine, Linköping University, SE-58185 Linköping, Sweden.<br /><searchLink fieldCode="AU" term="%22Nyman+E%22">Nyman E</searchLink>; Department of Biomedical Engineering, Linköping University, SE-58185 Linköping, Sweden.<br /><searchLink fieldCode="AU" term="%22Brännmark+C%22">Brännmark C</searchLink>; Department of Neuroscience and Physiology, Metabolic Physiology Section, University of Gothenburg, SE-41390 Göteborg, Sweden.<br /><searchLink fieldCode="AU" term="%22Olofsson+CS%22">Olofsson CS</searchLink>; Department of Neuroscience and Physiology, Metabolic Physiology Section, University of Gothenburg, SE-41390 Göteborg, Sweden.<br /><searchLink fieldCode="AU" term="%22Strålfors+P%22">Strålfors P</searchLink>; Department of Clinical and Experimental Medicine, Linköping University, SE-58185 Linköping, Sweden peter.stralfors@liu.se.
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  Data: <searchLink fieldCode="JN" term="%222984726R%22">The Biochemical journal</searchLink> [Biochem J] 2018 May 31; Vol. 475 (10), pp. 1807-1820. <i>Date of Electronic Publication: </i>2018 May 31.
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  Data: <i>Publisher: </i><searchLink fieldCode="PB" term="%22Published+by+Portland+Press+on+behalf+of+the+Biochemical+Society%22">Published by Portland Press on behalf of the Biochemical Society </searchLink><i>Country of Publication: </i>England <i>NLM ID: </i>2984726R <i>Publication Model: </i>Electronic <i>Cited Medium: </i>Internet <i>ISSN: </i>1470-8728 (Electronic) <i>Linking ISSN: </i><searchLink fieldCode="IS" term="%2202646021%22">02646021 </searchLink><i>NLM ISO Abbreviation: </i>Biochem J <i>Subsets: </i>MEDLINE
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        Value: 10.1042/BCJ20180144
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