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Signalling through Src family kinase isoforms is not redundant in models of thrombo-inflammatory vascular disease.

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Title: Signalling through Src family kinase isoforms is not redundant in models of thrombo-inflammatory vascular disease.
Authors: Harrison MJ; Institute of Cardiovascular Sciences, College of Medical and Dental Science, The Medical School, University of Birmingham, Birmingham, UK., Chimen M; Institute of Cardiovascular Sciences, College of Medical and Dental Science, The Medical School, University of Birmingham, Birmingham, UK., Hussain M; Institute of Cardiovascular Sciences, College of Medical and Dental Science, The Medical School, University of Birmingham, Birmingham, UK., Iqbal AJ; Institute of Cardiovascular Sciences, College of Medical and Dental Science, The Medical School, University of Birmingham, Birmingham, UK., Senis YA; Institute of Cardiovascular Sciences, College of Medical and Dental Science, The Medical School, University of Birmingham, Birmingham, UK., Nash GB; Institute of Cardiovascular Sciences, College of Medical and Dental Science, The Medical School, University of Birmingham, Birmingham, UK., Watson SP; Institute of Cardiovascular Sciences, College of Medical and Dental Science, The Medical School, University of Birmingham, Birmingham, UK., Rainger GE; Institute of Cardiovascular Sciences, College of Medical and Dental Science, The Medical School, University of Birmingham, Birmingham, UK.
Source: Journal of cellular and molecular medicine [J Cell Mol Med] 2018 Sep; Vol. 22 (9), pp. 4317-4327. Date of Electronic Publication: 2018 Jul 04.
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
Journal Info: Publisher: Wiley-Blackwell Country of Publication: England NLM ID: 101083777 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1582-4934 (Electronic) Linking ISSN: 15821838 NLM ISO Abbreviation: J Cell Mol Med Subsets: MEDLINE
Database: MEDLINE Ultimate
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  Data: Signalling through Src family kinase isoforms is not redundant in models of thrombo-inflammatory vascular disease.
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  Data: <searchLink fieldCode="AU" term="%22Harrison+MJ%22">Harrison MJ</searchLink>; Institute of Cardiovascular Sciences, College of Medical and Dental Science, The Medical School, University of Birmingham, Birmingham, UK.<br /><searchLink fieldCode="AU" term="%22Chimen+M%22">Chimen M</searchLink>; Institute of Cardiovascular Sciences, College of Medical and Dental Science, The Medical School, University of Birmingham, Birmingham, UK.<br /><searchLink fieldCode="AU" term="%22Hussain+M%22">Hussain M</searchLink>; Institute of Cardiovascular Sciences, College of Medical and Dental Science, The Medical School, University of Birmingham, Birmingham, UK.<br /><searchLink fieldCode="AU" term="%22Iqbal+AJ%22">Iqbal AJ</searchLink>; Institute of Cardiovascular Sciences, College of Medical and Dental Science, The Medical School, University of Birmingham, Birmingham, UK.<br /><searchLink fieldCode="AU" term="%22Senis+YA%22">Senis YA</searchLink>; Institute of Cardiovascular Sciences, College of Medical and Dental Science, The Medical School, University of Birmingham, Birmingham, UK.<br /><searchLink fieldCode="AU" term="%22Nash+GB%22">Nash GB</searchLink>; Institute of Cardiovascular Sciences, College of Medical and Dental Science, The Medical School, University of Birmingham, Birmingham, UK.<br /><searchLink fieldCode="AU" term="%22Watson+SP%22">Watson SP</searchLink>; Institute of Cardiovascular Sciences, College of Medical and Dental Science, The Medical School, University of Birmingham, Birmingham, UK.<br /><searchLink fieldCode="AU" term="%22Rainger+GE%22">Rainger GE</searchLink>; Institute of Cardiovascular Sciences, College of Medical and Dental Science, The Medical School, University of Birmingham, Birmingham, UK.
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        Value: 10.1111/jcmm.13721
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      – TitleFull: Signalling through Src family kinase isoforms is not redundant in models of thrombo-inflammatory vascular disease.
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              Text: 2018 Sep
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