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A dual role of miR-22 modulated by RelA/p65 in resensitizing fulvestrant-resistant breast cancer cells to fulvestrant by targeting FOXP1 and HDAC4 and constitutive acetylation of p53 at Lys382.

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Title: A dual role of miR-22 modulated by RelA/p65 in resensitizing fulvestrant-resistant breast cancer cells to fulvestrant by targeting FOXP1 and HDAC4 and constitutive acetylation of p53 at Lys382.
Authors: Wang B; Department of Biological Sciences, University of Lethbridge, Lethbridge, AB, Canada., Li D; Department of Biological Sciences, University of Lethbridge, Lethbridge, AB, Canada., Filkowski J; Department of Biological Sciences, University of Lethbridge, Lethbridge, AB, Canada., Rodriguez-Juarez R; Department of Biological Sciences, University of Lethbridge, Lethbridge, AB, Canada., Storozynsky Q; Department of Biological Sciences, University of Lethbridge, Lethbridge, AB, Canada., Malach M; Department of Biological Sciences, University of Lethbridge, Lethbridge, AB, Canada., Carpenter E; Department of Biological Sciences, University of Lethbridge, Lethbridge, AB, Canada., Kovalchuk O; Department of Biological Sciences, University of Lethbridge, Lethbridge, AB, Canada. olga.kovalchuk@uleth.ca.
Source: Oncogenesis [Oncogenesis] 2018 Jul 30; Vol. 7 (7), pp. 54. Date of Electronic Publication: 2018 Jul 30.
Publication Type: Journal Article
Journal Info: Publisher: Nature Pub. Group Country of Publication: United States NLM ID: 101580004 Publication Model: Electronic Cited Medium: Print ISSN: 2157-9024 (Print) Linking ISSN: 21579024 NLM ISO Abbreviation: Oncogenesis Subsets: PubMed not MEDLINE
Database: MEDLINE Ultimate
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  Data: A dual role of miR-22 modulated by RelA/p65 in resensitizing fulvestrant-resistant breast cancer cells to fulvestrant by targeting FOXP1 and HDAC4 and constitutive acetylation of p53 at Lys382.
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  Data: <searchLink fieldCode="AU" term="%22Wang+B%22">Wang B</searchLink>; Department of Biological Sciences, University of Lethbridge, Lethbridge, AB, Canada.<br /><searchLink fieldCode="AU" term="%22Li+D%22">Li D</searchLink>; Department of Biological Sciences, University of Lethbridge, Lethbridge, AB, Canada.<br /><searchLink fieldCode="AU" term="%22Filkowski+J%22">Filkowski J</searchLink>; Department of Biological Sciences, University of Lethbridge, Lethbridge, AB, Canada.<br /><searchLink fieldCode="AU" term="%22Rodriguez-Juarez+R%22">Rodriguez-Juarez R</searchLink>; Department of Biological Sciences, University of Lethbridge, Lethbridge, AB, Canada.<br /><searchLink fieldCode="AU" term="%22Storozynsky+Q%22">Storozynsky Q</searchLink>; Department of Biological Sciences, University of Lethbridge, Lethbridge, AB, Canada.<br /><searchLink fieldCode="AU" term="%22Malach+M%22">Malach M</searchLink>; Department of Biological Sciences, University of Lethbridge, Lethbridge, AB, Canada.<br /><searchLink fieldCode="AU" term="%22Carpenter+E%22">Carpenter E</searchLink>; Department of Biological Sciences, University of Lethbridge, Lethbridge, AB, Canada.<br /><searchLink fieldCode="AU" term="%22Kovalchuk+O%22">Kovalchuk O</searchLink>; Department of Biological Sciences, University of Lethbridge, Lethbridge, AB, Canada. olga.kovalchuk@uleth.ca.
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  Data: <searchLink fieldCode="JN" term="%22101580004%22">Oncogenesis</searchLink> [Oncogenesis] 2018 Jul 30; Vol. 7 (7), pp. 54. <i>Date of Electronic Publication: </i>2018 Jul 30.
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