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Frontline Science: TNF-α and GM-CSF1 priming augments the role of SOS1/2 in driving activation of Ras, PI3K-γ, and neutrophil proinflammatory responses.

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Title: Frontline Science: TNF-α and GM-CSF1 priming augments the role of SOS1/2 in driving activation of Ras, PI3K-γ, and neutrophil proinflammatory responses.
Authors: Suire S; Inositide Laboratory, The Babraham Institute, Cambridge, UK., Baltanas FC; Centro de Investigación del Cáncer-Instituto de Biología Molecular y Celular del Cáncer, (CSIC- Universitad de Salamanca) and CiberONC, Salamanca, Spain., Segonds-Pichon A; Bioinformatics Group, The Babraham Institute, Cambridge, UK., Davidson K; Inositide Laboratory, The Babraham Institute, Cambridge, UK., Santos E; Centro de Investigación del Cáncer-Instituto de Biología Molecular y Celular del Cáncer, (CSIC- Universitad de Salamanca) and CiberONC, Salamanca, Spain., Hawkins PT; Inositide Laboratory, The Babraham Institute, Cambridge, UK., Stephens LR; Inositide Laboratory, The Babraham Institute, Cambridge, UK.
Source: Journal of leukocyte biology [J Leukoc Biol] 2019 Oct; Vol. 106 (4), pp. 815-822. Date of Electronic Publication: 2019 Feb 05.
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
Journal Info: Publisher: Oxford University Press Country of Publication: England NLM ID: 8405628 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1938-3673 (Electronic) Linking ISSN: 07415400 NLM ISO Abbreviation: J Leukoc Biol Subsets: MEDLINE
Database: MEDLINE Ultimate
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  Data: Frontline Science: TNF-α and GM-CSF1 priming augments the role of SOS1/2 in driving activation of Ras, PI3K-γ, and neutrophil proinflammatory responses.
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  Data: <searchLink fieldCode="AU" term="%22Suire+S%22">Suire S</searchLink>; Inositide Laboratory, The Babraham Institute, Cambridge, UK.<br /><searchLink fieldCode="AU" term="%22Baltanas+FC%22">Baltanas FC</searchLink>; Centro de Investigación del Cáncer-Instituto de Biología Molecular y Celular del Cáncer, (CSIC- Universitad de Salamanca) and CiberONC, Salamanca, Spain.<br /><searchLink fieldCode="AU" term="%22Segonds-Pichon+A%22">Segonds-Pichon A</searchLink>; Bioinformatics Group, The Babraham Institute, Cambridge, UK.<br /><searchLink fieldCode="AU" term="%22Davidson+K%22">Davidson K</searchLink>; Inositide Laboratory, The Babraham Institute, Cambridge, UK.<br /><searchLink fieldCode="AU" term="%22Santos+E%22">Santos E</searchLink>; Centro de Investigación del Cáncer-Instituto de Biología Molecular y Celular del Cáncer, (CSIC- Universitad de Salamanca) and CiberONC, Salamanca, Spain.<br /><searchLink fieldCode="AU" term="%22Hawkins+PT%22">Hawkins PT</searchLink>; Inositide Laboratory, The Babraham Institute, Cambridge, UK.<br /><searchLink fieldCode="AU" term="%22Stephens+LR%22">Stephens LR</searchLink>; Inositide Laboratory, The Babraham Institute, Cambridge, UK.
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  Data: <searchLink fieldCode="JN" term="%228405628%22">Journal of leukocyte biology</searchLink> [J Leukoc Biol] 2019 Oct; Vol. 106 (4), pp. 815-822. <i>Date of Electronic Publication: </i>2019 Feb 05.
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  Data: <i>Publisher: </i><searchLink fieldCode="PB" term="%22Oxford+University+Press%22">Oxford University Press </searchLink><i>Country of Publication: </i>England <i>NLM ID: </i>8405628 <i>Publication Model: </i>Print-Electronic <i>Cited Medium: </i>Internet <i>ISSN: </i>1938-3673 (Electronic) <i>Linking ISSN: </i><searchLink fieldCode="IS" term="%2207415400%22">07415400 </searchLink><i>NLM ISO Abbreviation: </i>J Leukoc Biol <i>Subsets: </i>MEDLINE
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        Value: 10.1002/JLB.2HI0918-359RR
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              Text: 2019 Oct
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