Imbalanced sphingolipid signaling is maintained as a core proponent of a cancerous phenotype in spite of metabolic pressure and epigenetic drift.
Saved in:
| Title: | Imbalanced sphingolipid signaling is maintained as a core proponent of a cancerous phenotype in spite of metabolic pressure and epigenetic drift. |
|---|---|
| Authors: | Speirs MMP; Department of Chemistry and Biochemistry, Brigham Young University, Provo, Utah, USA., Swensen AC; Department of Chemistry and Biochemistry, Brigham Young University, Provo, Utah, USA., Chan TY; Department of Chemistry and Biochemistry, Brigham Young University, Provo, Utah, USA., Jones PM; Department of Chemistry and Biochemistry, Brigham Young University, Provo, Utah, USA., Holman JC; Department of Chemistry and Biochemistry, Brigham Young University, Provo, Utah, USA., Harris MB; Department of Chemistry and Biochemistry, Brigham Young University, Provo, Utah, USA., Maschek JA; Health Sciences Cores-Metabolomics, University of Utah, Salt Lake, Utah, USA., Cox JE; Health Sciences Cores-Metabolomics, University of Utah, Salt Lake, Utah, USA., Carson RH; Department of Chemistry and Biochemistry, Brigham Young University, Provo, Utah, USA., Hill JT; Department of Physiology and Developmental Biology, Brigham Young University, Provo, Utah, USA., Andersen JL; Department of Chemistry and Biochemistry, Brigham Young University, Provo, Utah, USA., Prince JT; Department of Chemistry and Biochemistry, Brigham Young University, Provo, Utah, USA., Price JC; Department of Chemistry and Biochemistry, Brigham Young University, Provo, Utah, USA. |
| Source: | Oncotarget [Oncotarget] 2019 Jan 11; Vol. 10 (4), pp. 449-479. Date of Electronic Publication: 2019 Jan 11 (Print Publication: 2019). |
| Publication Type: | Journal Article |
| Journal Info: | Publisher: Impact Journals Country of Publication: United States NLM ID: 101532965 Publication Model: eCollection Cited Medium: Internet ISSN: 1949-2553 (Electronic) Linking ISSN: 19492553 NLM ISO Abbreviation: Oncotarget Subsets: PubMed not MEDLINE |
| Database: | MEDLINE Ultimate |
Be the first to leave a comment!