mTORC1 underlies age-related muscle fiber damage and loss by inducing oxidative stress and catabolism.

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Bibliographic Details
Title: mTORC1 underlies age-related muscle fiber damage and loss by inducing oxidative stress and catabolism.
Authors: Tang H; Division of Thoracic Surgery, Department of Cardiothoracic Surgery, Stanford University School of Medicine, Stanford, California.; VA Palo Alto Healthcare System, Palo Alto, California., Inoki K; Life Science Institute, University of Michigan, Ann Arbor, Michigan.; Department of Molecular and Integrative Physiology, University of Michigan, Ann Arbor, Michigan., Brooks SV; Department of Molecular and Integrative Physiology, University of Michigan, Ann Arbor, Michigan., Okazawa H; Department of Pharmacology and Moores Cancer Center, University of California San Diego, La Jolla, California., Lee M; Division of Thoracic Surgery, Department of Cardiothoracic Surgery, Stanford University School of Medicine, Stanford, California.; VA Palo Alto Healthcare System, Palo Alto, California., Wang J; Life Science Institute, University of Michigan, Ann Arbor, Michigan., Kim M; Division of Thoracic Surgery, Department of Cardiothoracic Surgery, Stanford University School of Medicine, Stanford, California.; VA Palo Alto Healthcare System, Palo Alto, California., Kennedy CL; Division of Thoracic Surgery, Department of Cardiothoracic Surgery, Stanford University School of Medicine, Stanford, California.; VA Palo Alto Healthcare System, Palo Alto, California., Macpherson PCD; Molecular and Behavioral Neuroscience Institute and Department of Biological Chemistry, University of Michigan, Ann Arbor, Michigan., Ji X; Human Immune Monitoring Center, Stanford University School of Medicine, Stanford, California., Van Roekel S; Department of Pathology and Geriatrics Center, University of Michigan, Ann Arbor, Michigan., Fraga DA; Division of Thoracic Surgery, Department of Cardiothoracic Surgery, Stanford University School of Medicine, Stanford, California.; VA Palo Alto Healthcare System, Palo Alto, California., Wang K; Division of Thoracic Surgery, Department of Cardiothoracic Surgery, Stanford University School of Medicine, Stanford, California.; VA Palo Alto Healthcare System, Palo Alto, California., Zhu J; Division of Thoracic Surgery, Department of Cardiothoracic Surgery, Stanford University School of Medicine, Stanford, California.; VA Palo Alto Healthcare System, Palo Alto, California., Wang Y; Division of Thoracic Surgery, Department of Cardiothoracic Surgery, Stanford University School of Medicine, Stanford, California.; VA Palo Alto Healthcare System, Palo Alto, California., Sharp ZD; Department of Molecular Medicine, University of Texas Health Science Center at San Antonio, San Antonio, Texas., Miller RA; Department of Pathology and Geriatrics Center, University of Michigan, Ann Arbor, Michigan., Rando TA; VA Palo Alto Healthcare System, Palo Alto, California.; Paul F. Glenn Laboratories for the Biology of Aging and Department of Neurology and Neurological Sciences, Stanford University School of Medicine, Stanford, California., Goldman D; Molecular and Behavioral Neuroscience Institute and Department of Biological Chemistry, University of Michigan, Ann Arbor, Michigan., Guan KL; Department of Pharmacology and Moores Cancer Center, University of California San Diego, La Jolla, California., Shrager JB; Division of Thoracic Surgery, Department of Cardiothoracic Surgery, Stanford University School of Medicine, Stanford, California.; VA Palo Alto Healthcare System, Palo Alto, California.
Source: Aging cell [Aging Cell] 2019 Jun; Vol. 18 (3), pp. e12943. Date of Electronic Publication: 2019 Mar 29.
Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, U.S. Gov't, Non-P.H.S.
Journal Info: Publisher: Wiley-Blackwell Country of Publication: England NLM ID: 101130839 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1474-9726 (Electronic) Linking ISSN: 14749718 NLM ISO Abbreviation: Aging Cell Subsets: MEDLINE
Database: MEDLINE Ultimate
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